Determination of Left Ventricular Volumes by Ultrasound

Fortuin, Nicholas J.; Hood, William P.; Sherman, Mark E.; Craige, Ernest

doi:10.1161/01.cir.44.4.575

Cited by 280 publications

(56 citation statements)

References 24 publications

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“…In support of the ultrasonic approach for measuring LV chamber dimensions, it should be pointed out that ultrasonic techniques have been extensively utilized to measure LV dimensions in experimental animals (33) and in man (17)(18)(19)(20)(21)(22)(23). Recently, Ratshin, Rackley, and Russell have reported (34) on the use of a combined hemodynamic and ultrasonic technique, similar to that used in our study, for the measurement of LV wall stress.…”

Section: Resultsmentioning

confidence: 99%

“…At the time of cardiac catheterization, all patients were well compensated with regard to congestive heart failure, with the exception of volume overload patients 4, 7, and 12, whose data are given in Table I (13)(14)(15)(16). The use of pulsed reflected ultrasound to measure LV dimensions and wall thickness in man has been validated now by several investigators (17)(18)(19)(20)(21)(22)(23), and is discussed in detail in the Discussion section. In this study, the LV internal diameter, D, was measured as the vertical distance between echoes from the endocardial surfaces of the interventricular septum and LV posterior wall, and posterior wall thickness, h, was measured as the distance between endocardial and epicardial echoes on theultrasonic tracing (Fig.…”

Section: Introductionmentioning

confidence: 99%

See 1 more Smart Citation

Wall stress and patterns of hypertrophy in the human left ventricle.

Grossman¹,

Jones²,

McLaurin³

1975

J. Clin. Invest.

2,190

981

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Section: Resultsmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Wall stress and patterns of hypertrophy in the human left ventricle.

Grossman¹,

Jones²,

McLaurin³

1975

J. Clin. Invest.

2,190

981

View full text Add to dashboard Cite

“…ventricular function,9 10 and there is preliminary data suggesting that it has promise in assessing global left ventricular performance." 16 We compared EF and ventricular volumes obtained from TDE images with corresponding determinations derived from RVGs and from x-ray cineangiograms made at cardiac catheterization in the same series of patients.…”

Section: Discussionmentioning

confidence: 99%

Acute hemodynamic effects of cigarette smoking in man assessed by systolic time intervals and echocardiography.

Rabinowitz¹,

Thorp²,

Huber³

et al. 1979

Circulation

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SUMMARY Sixteen healthy subjects, ages 18-35 years, were studied in the supine position by means of systolic time intervals and echocardiography before and after smoking a high-nicotine cigarette (2.5 mg nicotine) and a tobacco cigarette of very low nicotine content (< 0.02 mg nicotine) to assess and compare the immediate effects upon left ventricular function.Smokers (n = 12) and nonsmokers (n = 4) behaved alike. High-and low-nicotine cigarettes both caused significant increases in heart rate, systolic and diastolic blood pressure and the triple product (systolic blood pressure X left ventricular ejection (LVET) X heart rate), prolonged LVETc and decreased the preejection period (PEP) and PEP/LVET. In addition, smoking a nicotine reference cigarette increased the echocardiographically derived LV end-diastolic volume by 7.5%, augmented ejection fraction by 4%, while significantly enhancing mean normalized circumferential fiber shortening by 12.5% and mean normalized posterior wall velocity by 9%. Smoking a tobacco cigarette of ultra-low nicotine content resulted in comparable increases in ejection fraction and mean circumferential fiber shortening, albeit on the basis of a significant decrease in end-systolic volume without alteration in end-diastolic volume.These data suggest that in the supine position smoking a high-nicotine cigarette acutely increases venous return and augments the principal determinants of myocardial oxygen consumption heart rate, contractility, preload and afterload and that cigarette smoke may contain inotropic and chronotropic substances other than nicotine.CIGARETTE SMOKING has been linked epidemiologically as a risk factor in the development of myocardial infarction and has been associated with an increase in sudden death among chronic cigarette consumers.', I The cause of this increased morbidity and mortality has not been established; among the factors under consideration are hypoxemia,3 enhanced cor-

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“…In the present series, wall thickening above the normal range was present in 95% of patients. Wall thickness averaged 16 In contrast, the internal dimensions of the left ventricle in diastole and systole were increased above normal in only 25% of patients. Moreover, marked ventricular dilatation (i.e., end-diastolic dimension greater than 50% above normal) was present in only one patient, whose left ventricular end-diastolic dimension was 86 mm and who also had coronary artery disease and a history of very heavy alcohol intake for several years before operation.…”

Section: Discussionmentioning

confidence: 80%

Evaluation of aortic valve replacement in patients with valvular aortic stenosis.

et al. 1980

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Echocardiographic and hemodynamic studies were obtained in 42 consecutive patients undergoing aortic valve replacement for isolated aortic stenosis. Concentric left ventricular (LV) wall thickening, the most common preoperative abnormality, occurred in 95% of patients. LV dilation with reduced fractional shortening was noted in approximately 25% of patients but was severe in only one patient. Six months after operation, LV wall thickness had decreased on average but had not returned to normal and fractional shortening was unchanged. Repeat measurements in 13 patients an average of 37 months after operation were unchanged compared with measurements made 6 months after operation. When patients were subdivided into those with LV dilatation and those without, we found that patients with dilated ventricles preoperatively had a greater decrease in LV internal dimension and mass than those without preoperative dilatation. The patient data also were examined for possible association with mortality. One operative (2%) and five late cardiac (13%) deaths occurred. No preoperative or 6-month postoperative echocardiographic or hemodynamic measurement was strongly associated with these deaths, nor were any late deaths due to congestive heart failure. Compared with preoperative measurements in symptomatic patients who were operated for isolated aortic regurgitation, patients with aortic stenosis had smaller left ventricles with less depression of systolic function, as well as less aortic root and left atrial dilatation. Our data do not support the concept that the aortic valve should be replaced before the onset of symptoms to prevent irreversible LV damage in patients with isolated aortic stenosis.

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Determination of Left Ventricular Volumes by Ultrasound

Abstract: SUMMARYWe compared dimensions of the left ventricular minor axis (S) measured at end-

Cited by 280 publications

References 24 publications

Wall stress and patterns of hypertrophy in the human left ventricle.

Wall stress and patterns of hypertrophy in the human left ventricle.

Acute hemodynamic effects of cigarette smoking in man assessed by systolic time intervals and echocardiography.

Evaluation of aortic valve replacement in patients with valvular aortic stenosis.

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