Dependence of Erythroblast Proliferation and Differentiation Manner in Mice Hemopoietic Tissue Regeneration on Lymphoid-Macrophagal System Cells

Бабаева, А. Г.; Belan, E. I.

doi:10.1007/978-1-4684-9057-2_12

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“…Plenum Publishing Corporation were isolated 4 h after bleeding as described previously [1] and enriched with macrophages by complement-dependent lysis of T cells using murine antimurine anti-Thy-l.2 monoclonal antibodies (Cedarlane, Canada) (1:100) and rabbit serum as complement (1:10). The indicated dilutions of the serum and monoclonal antibodies were optimal and caused the death of 89.3% thymocytes.…”

Section: Methodsmentioning

confidence: 99%

“…A transito13" increase in the content of basophilic erythroblasts (BE) in the bone marrow paralleled by a decrease of their mitotic activity may be considered as a bone marrow marker of "reserve erythropoiesis" [3,7]. Moreover, blood loss enhances polychromatophilic erythroblasts (PE) amplification and initiates mitoses in oxyphilic erythroblasts (OE), which normally almost never divide [1,3]. …”

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confidence: 99%

“…Under conditions of massive blood loss resident peritoneal ceils (PC) acquire the ability to trigger stress erythropoiesis in normal syngeneic recipients during the adoptive transfer of these cells [1,4]. Their effect on recipient erythropoiesis manifests itself by activation of various routes of terminal erythroid differentiation characteristic of anemic animals.…”

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The role of peritoneal T lymphocytes and macrophages in the initiation of stress erythropoiesisin vivo following a single massive blood loss in mice

Belan

Головкина

1994

Bull Exp Biol Med

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Peritoneal ceils of anemic donors enriched with macrophages at the expense of Tcell lysis using anti-Thy-1.2 monoclonal antibodies are capable of triggering various routes of terminal erythroid differentiation characteristic of stress erythropoiesis during their adoptive transfer to normal syngeneic recipients. Stimulation of the proliferation of polychromatophilic erythroblasts and initiation of mitoses in oxyphilic erythroblasts are regulated by interacting T ceUs and macrophages, and the "reserve erythropoiesis" mainly by macrophages. Key Words: stress erythropoiesis; peritoneal cells; reserve erythropoiesis; blood lossIn mammals with severe anemias the erythroid precursors, on reaching the terminal stages, form red ceils as a result of terminal differentiation by various routes. One of these routes, "reserve erythropoiesis," is an emergency route of accelerated differentiation of ceils providing for the survival of the organism during the early periods after massive blood loss. It consists in the omission of severn terminal stages and the rapid release of morphologically immature cells into the blood [2,3,[6][7][8]. A transito13" increase in the content of basophilic erythroblasts (BE) in the bone marrow paralleled by a decrease of their mitotic activity may be considered as a bone marrow marker of "reserve erythropoiesis" [3,7]. Moreover, blood loss enhances polychromatophilic erythroblasts (PE) amplification and initiates mitoses in oxyphilic erythroblasts (OE), which normally almost never divide [1,3].

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Section: Methodsmentioning

confidence: 99%

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