2018
DOI: 10.3758/s13415-018-0597-1
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Delay discounting mediates the association between posterior insular cortex volume and social media addiction symptoms

Abstract: Addiction-like symptoms in relation to excessive and compulsive social media use are common in the general population. Because they can lead to various adverse effects, there is a growing need to understand the brain systems and processes that are involved in potential social media addiction. We focus on the morphology of the posterior subdivision of the insular cortex (i.e., the insula), because it has been shown to be instrumental to supporting the maintenance of substance addictions and problematic behavior… Show more

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Cited by 61 publications
(47 citation statements)
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“…Impairments in general executive functions and especially specific inhibitory control are assumed to occur in later stages of the addiction process 24 , which may explain the missing direct associations in the current sample including individuals who are at most in an early stage or at risk of developing social-networks-use disorder. This is in line with neuroscientific evidence that disordered social media use symptoms in non-clinical samples are typically not associated with prefrontal brain impairments, which would manifest in reduced executive function and inhibition 45,[54][55][56]81 . Future research should examine when and how prefrontal brain impairment, reduced executive function, and inhibition abilities contribute to the transition of impulsive non-clinical populations into significant impairments to normal functioning that can be classified as social-networks-use disorder cases.…”
Section: Discussionsupporting
confidence: 86%
See 1 more Smart Citation
“…Impairments in general executive functions and especially specific inhibitory control are assumed to occur in later stages of the addiction process 24 , which may explain the missing direct associations in the current sample including individuals who are at most in an early stage or at risk of developing social-networks-use disorder. This is in line with neuroscientific evidence that disordered social media use symptoms in non-clinical samples are typically not associated with prefrontal brain impairments, which would manifest in reduced executive function and inhibition 45,[54][55][56]81 . Future research should examine when and how prefrontal brain impairment, reduced executive function, and inhibition abilities contribute to the transition of impulsive non-clinical populations into significant impairments to normal functioning that can be classified as social-networks-use disorder cases.…”
Section: Discussionsupporting
confidence: 86%
“…Together, the findings emphasize that the presentation of specific Internet-related stimuli affects attentional processes, which could result in diminished control over the use of Internet/social-networks, if accompanied by high attentional impulsivity as a predisposing variable. This view has also been supported from a neuroscientific standpoint, by studies showing that activation and morphology of centers of impulsivity and diminished ability to overcome impulsions are associated with social-networks-use disorder symptom severity 45,56 .…”
Section: Discussionmentioning
confidence: 92%
“…This interoceptive awareness system integrates homeostatic signals, thereby regulating processes of the dual-process system ( 24 ) and subserving behaviors through the subjective feelings of urges /cravings that it mediates ( 25 ). Evidence has been accumulating regarding the viability of the triadic system perspective for explaining excessive behaviors ( 26 , 27 ), as well as regarding the role of the interoceptive awareness system in decisional deficits ( 28 )…”
Section: The Triadic Neurocognitive Systemmentioning
confidence: 99%
“…, 2014 ), hippocampus ( Lebreton et al. , 2013 ), insula ( Turel et al. , 2018 ) as well as temporal pole and temporoparietal junction ( Pehlivanova et al.…”
Section: Introductionmentioning
confidence: 99%