Degradation of 1-naphthylamine by a UV enhanced Fe2+/peroxymonosulfate system: A novel pH-dependent activation pathway

Liu, Jianghui; So, Hiu Lam; Chu, Wei

doi:10.1016/j.cej.2022.136299

Cited by 8 publications

(2 citation statements)

References 79 publications

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“…Then, part of these iron oxides are reduced to Fe 2+ under high-temperature conditions, leading to an Fe 2+ and Fe 3+ mixture on the TiO 2 surface. 45 Simultaneously, OH − ions from the HT solution chemically dissolve the TiO 2 surface, resulting in the formation of HTiO 3 − . 40 Ca 2+ , Fe 2+ , and Fe 3+ ions react with SiO 3 2− and HTiO 3 − , forming Fe-doped Ca(TiSiO 5 ) nuclei, and with the prolonged HT process they grow rapidly to nanosword-like nanoflakes due to the minimized surface energy according to the Ostwald ripening process.…”

Section: Resultsmentioning

confidence: 99%

“…During the HT process, partial Fe 3+ ions in TiO 2 migrate to the outer surface and react with H 2 O, forming iron oxides Fe III (OH) 2+ . Then, part of these iron oxides are reduced to Fe 2+ under high-temperature conditions, leading to an Fe 2+ and Fe 3+ mixture on the TiO 2 surface . Simultaneously, OH – ions from the HT solution chemically dissolve the TiO 2 surface, resulting in the formation of HTiO 3 – .…”

Section: Resultsmentioning

confidence: 99%

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Alkaline “Nanoswords” Coordinate Ferroptosis-like Bacterial Death for Antibiosis and Osseointegration

et al. 2023

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Ferroptosis is an iron-dependent cell death and is associated with cancer therapy. Can it play a role in resistance of postoperative infection of implants, especially with an extracellular supplement of Fe ions in a non-cytotoxic dose? To answer this, "nanoswords" of Fe-doped titanite are fabricated on a Ti implant surface to resist bacterial invasion by a synergistic action of ferroptosis-like bacteria killing, proton disturbance, and physical puncture. The related antibiosis mechanism is explored by atomic force microscopy and genome sequencing. The nanoswords induce an increased local pH value, which not only weakens the proton motive force, reducing adenosine triphosphate synthesis of Staphylococcus aureus, but also decreases the membrane modulus, making the nanoswords distort and even puncture a bacterial membrane easily. Simultaneously, more Fe ions are taken by bacteria due to increased bacterial membrane permeability, resulting in ferroptosis-like death of bacteria, and this is demonstrated by intracellular iron enrichment, lipid peroxidation, and glutathione depletion. Interestingly, a microenvironment constructed by these nanoswords improves osteoblast behavior in vitro and bone regeneration in vivo. Overall, the nanoswords can induce ferroptosis-like bacterial death without cytotoxicity and have great promise in applications with clinical implants for outstanding antibiosis and biointegration performance.

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Section: Resultsmentioning

confidence: 99%

Section: Resultsmentioning

confidence: 99%