Decreasing frequency of anesthetic cardiac arrests

Keenan, Richard L.; Boyan, C. Paul

doi:10.1016/0952-8180(91)90174-l

Cited by 87 publications

(30 citation statements)

References 8 publications

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“…Cardiac arrests are identifiable and the immediate causative factors are usually able to be determined 36 . Anaesthetic cardiac arrest rate has decreased significantly over time from 2.1 per 10,000 (1969-1978) to 1.0 per 10,000 (1979)(1980)(1981)(1982)(1983)(1984)(1985)(1986)(1987)(1988) 37 . Much of this decrease in the incidence is due to a greater decrease in preventable arrests due to respiratory causes 37 .…”

Section: Cardiac Arrestmentioning

confidence: 99%

Measuring Anaesthetic Outcomes

Lee

Lum

1996

Anaesth Intensive Care

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Section: Cardiac Arrestmentioning

confidence: 99%

Measuring Anaesthetic Outcomes

Lee

Lum

1996

Anaesth Intensive Care

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“…However, there is substantial evidence that it reduces the risks of incidents and accidents both by detecting the consequences of errors, and by giving early warning that the condition of a patient is deteriorating 5, 6, 7, 8, 9, 10, 11.…”

Section: Introductionmentioning

confidence: 99%

Recommendations for standards of monitoring during anaesthesia and recovery 2015 : Association of Anaesthetists of Great Britain and Ireland

Checketts¹,

et al. 2015

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SummaryThis guideline updates and replaces the 4th edition of the AAGBI Standards of Monitoring published in 2007. The aim of this document is to provide guidance on the minimum standards for physiological monitoring of any patient undergoing anaesthesia or sedation under the care of an anaesthetist. The recommendations are primarily aimed at anaesthetists practising in the United Kingdom and Ireland. Minimum standards for monitoring patients during anaesthesia and in the recovery phase are included. There is also guidance on monitoring patients undergoing sedation and also during transfer of anaesthetised or sedated patients. There are new sections discussing the role of monitoring depth of anaesthesia, neuromuscular blockade and cardiac output. The indications for end‐tidal carbon dioxide monitoring have been updated.

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“…Although there are no studies directly comparing the incidence of cardiac arrest in laparoscopic and ‘open’ surgery, the above mentioned numbers are slightly increased compared to the anesthetic intraoperative cardiac arrest rate of 10/100,000 calculated for general surgeries between 1979–1988 [15, 16]. Among the causes of cardiac arrest there are two particularly associated to laparoscopy: the profound vasovagal response to rapid peritoneal distension and gas embolism.…”

Section: Cardiovascular Systemmentioning

confidence: 99%

Circulatory and Respiratory Complications of Carbon Dioxide Insufflation

Gutt¹,

Oniu

Mehrabi³

et al. 2004

Dig Surg

300

272

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Background: Although providing excellent outcome results, laparoscopy also induces particular pathophysiological changes in response to pneumoperitoneum. Knowledge of the pathophysiology of a CO₂ pneumoperitoneum can help minimize complications while profiting from the benefits of laparoscopic surgery without concerns about its safety. Methods: A review of articles on the pathophysiological changes and complications of carbon dioxide pneumoperitoneum as well as prevention and treatment of these complications was performed using the Medline database. Results: The main pathophysiological changes during CO₂ pneumoperitoneum refer to the cardiovascular system and are mainly correlated with the amount of intra-abdominal pressure in combination with the patient’s position on the operating table. These changes are well tolerated even in older and more debilitated patients, and except for a slight increase in the incidence of cardiac arrhythmias, no other significant cardiovascular complications occur. Although there are important pulmonary pathophysiological changes, hypercarbia, hypoxemia and barotraumas, they would develop rarely since effective ventilation monitoring and techniques are applied. The alteration in splanchnic perfusion is proportional with the increase in intra-abdominal pressure and duration of pneumoperitoneum. Conclusion: A moderate-to-low intra-abdominal pressure (<12 mm Hg) can help limit the extent of the pathophysiological changes since consecutive organ dysfunctions are minimal, transient and do not influence the outcome.

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Decreasing frequency of anesthetic cardiac arrests

Cited by 87 publications

References 8 publications

Measuring Anaesthetic Outcomes

Measuring Anaesthetic Outcomes

Recommendations for standards of monitoring during anaesthesia and recovery 2015 : Association of Anaesthetists of Great Britain and Ireland

Circulatory and Respiratory Complications of Carbon Dioxide Insufflation

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