A 4-year-old Siberian Husky dog was treated with brown snake antivenom by his regular veterinarian after a witnessed episode of brown snake envenomation. The dog was discharged 5 hours post presentation despite an ongoing coagulopathy. The dog was presented to the emergency centre 2 hours later because the owner believed the dog to be in pain. Initial examination revealed an ambulatory but neurologically normal patient with thoracolumbar pain and laboratory evidence of a coagulopathy. Despite correction of the coagulopathy, the signs progressed to bilateral hind limb paresis after approximately 3 hours of hospitalisation, and continued to deteriorate over the next 56 hours to loss of deep pain perception in the right hind limb. Computed tomography imaging identified the presence of an extradural haematoma which was subsequently removed via a hemilaminectomy. Surgical decompression was successful in treating the spinal compression and the dog recovered with minimal complications. To our knowledge this is the first report of extradural haematoma secondary to coagulopathy induced by brown snake envenomation.Keywords brown snake; envenomation; coagulopathy; dog; elapid; extradural haematoma; Pseudonaja Abbreviations ACT, activated clotting time; CT, computed tomography; CTM, computed tomography with myelography; FFP, fresh frozen plasma; L2, second lumbar vertebra; L3, third lumbar vertebra; MRI, magnetic resonance imaging Envenomation is common in the warmer months and is typically associated with lower motor neuron paresis or paralysis and evidence of a coagulopathy without significant muscle damage.To our knowledge a confirmed case of compressive spinal disease in the dog has not been reported as a complication of brown snake envenomation. This case report describes the presentation, diagnosis and treatment of a dog that developed hind limb paralysis secondary to extradural haematoma formation following brown snake envenomation.A 4-year-old, male neutered Siberian Husky dog was presented to his local veterinarian after his owner observed him being bitten by a snake. The history detailed collapse followed by apparent recovery, approximately 10 minutes post envenomation (Figure 1), however ongoing hind limb weakness prompted presentation to the local veterinary practice. On presentation, examination revealed hindlimb paresis, ataxia and a marked coagulopathy with an activated clotting time (ACT) of greater than 300 s (normal < 120 s). Brown snake envenomation was confirmed on a urine sample using a snake venom detection kit test (CSL Ltd, Australia) and subsequent visual identification of the snake. Gross pigmenturia was also noted in the voided urine sample. Treatment was commenced with 500 U of brown snake antivenom (CSL Ltd, Australia) after premedication with 3 mg dexamethasone sodium phosphate (Troy Laboratories, Australia). Two and 4 h after antivenom administration, ACT was still greater than 300 s however the ataxia and hind limb weakness had resolved. No further antivenom was administered. Despite the ongo...