Abstract:In CBA mice infected with influenza viruses A/H1N1/California/04/2009 and A/H5N1/Goose/ Krasnoozerskoye/627/05 in a dose of 10 MLD 50 , the mechanisms of death of pulmonary alveolocytes over 10 postinfection days were studied by light microscopy, immunohistochemistry, and morphometry. In mice infected with A/H1N1, alveolocytes died predominantly via necrosis, while apoptosis mostly employed the mitochondrial pathway. In mice infected with A/ H5N1, apoptosis was the dominant mechanism of alveolocyte death proce… Show more
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