1976
DOI: 10.1007/bf01685893
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DDE Poisoning in wild great blue heron

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Cited by 17 publications
(4 citation statements)
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“…Using this relationship, theoretical hepatic concentrations of ΣDDT (DDT þDDE) in the species in this study would be 5.33 mg/g in American black vulture, 5.35 mg/g in Southern crested caracara and 8.70 mg/g in Turkey vulture. DDE levels of 124 mg/g in the livers of some bird species (Ardeidae) were associated with broken eggshell (Pratt, 1972), while levels of 569 mg/g were associated with deaths of birds (Call et al, 1976). These levels are much higher than those estimated in this study; however, we cannot discard sublethal effects since the values found are far from normal at least for these birds.…”
Section: Effect Assessmentcontrasting
confidence: 44%
“…Using this relationship, theoretical hepatic concentrations of ΣDDT (DDT þDDE) in the species in this study would be 5.33 mg/g in American black vulture, 5.35 mg/g in Southern crested caracara and 8.70 mg/g in Turkey vulture. DDE levels of 124 mg/g in the livers of some bird species (Ardeidae) were associated with broken eggshell (Pratt, 1972), while levels of 569 mg/g were associated with deaths of birds (Call et al, 1976). These levels are much higher than those estimated in this study; however, we cannot discard sublethal effects since the values found are far from normal at least for these birds.…”
Section: Effect Assessmentcontrasting
confidence: 44%
“…The Gray Heron apparently avoids the adverse effects of organochlorine residues by recycling and laying replacement clutches (Prestt 1970b), although residues and eggshell thickness were not defined in replacement clutches. The only recorded mortality of an adult Great Blue Heron from an organochlorine occurred in South Dakota where a bird died from DDE poisoning (Call et al 1976). The only recorded mortality of an adult Great Blue Heron from an organochlorine occurred in South Dakota where a bird died from DDE poisoning (Call et al 1976).…”
Section: Discussionmentioning
confidence: 99%
“…While one route of exposure to OCPs is occupational, OCPs are now pervasive environmental contaminants 77. The huge volume of use over the past 50 years in agricultural and home pest control has resulted in the contamination of soil, water, plant, and animal species,54, 73, 78–80 with environmental and food residue contamination leading to chronic low‐level exposure even among those who are not occupationally exposed 81, 82. This contamination has created a long‐lasting environmental problem, as the members of the organochlorine class of pesticides are resistant to degradation and have been labeled as “persistent bioaccumulators,” detectible in soils for 30 years after their application 73, 83…”
Section: Nature Vs Nurturementioning
confidence: 99%