Das Ilioinguinalis-Syndrom<sup>*</sup>

Mumenthaler, A; Mumenthaler, M; Luciani, Giovanni Battista; Kramer, John Paul

doi:10.1055/s-0028-1111468

Cited by 17 publications

(5 citation statements)

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“…TTS was first described in 1962 by Keck [71. The clinical features in our series are typical of TTS [3, [8][9][10]. The symptoms consisted of burning pain and paresthesia in the toes and along the sole of the foot.…”

Section: Discussionmentioning

confidence: 99%

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Tarsal tunnel syndrome: Electrophysiological study

Sarala

Kuba

et al. 1979

Annals of Neurology

125

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Tarsal tunnel syndrome (TTS) is a rare compression neuropathy of the posterior tibial nerve. Typical symptoms are burning pain and paresthesia in the toes and along the sole of the foot. The presence of Tinel's sign and objective sensory loss in the territory of any of the terminal branches of the posterior tibial nerve are diagnostically helpful. The terminal latency and sensory nerve conduction velocity in medial and lateral plantar nerves were studied in 20 normal controls and 21 cases of TTS in 17 patients. Prolonged terminal latency was observed in 11 cases, with TTS, while sensory nerve conduction abnormality (either absent nerve potential or slow sensory nerve conduction velocity) was found in 19. The sensory nerve conduction velocity in the lateral and medial plantar nerves is a superior objective diagnostic index of TTS.

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Section: Discussionmentioning

confidence: 99%

“…The treatment of 'ITS is surgical release of the flexor retinaculum and neurolysis of the medial and lateral plantar nerves [3, [8][9][10]. Complete relief of symptoms was achieved in 78 to 93% of the operative cases [8, …”

Section: Discussionmentioning

confidence: 99%

Tarsal tunnel syndrome: Electrophysiological study

Sarala

Kuba

et al. 1979

Annals of Neurology

125

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“…Palpation of pubic symphysis, 42 arches and tubercles Neurology Sensibility test, palpation for nerve entrapment [33][34][35][36] Palpation of the prostate, 37 38 39 scrotum, sacrotuberal ligament 40 and pelvic floor 41 Iliopsoas-related pain was the primary clinical entity in 73 (35%) patients. Other primary clinical entities were relatively rare.…”

Section: Region Diagnostic Methodsmentioning

confidence: 99%

Long-standing groin pain in sportspeople falls into three primary patterns, a “clinical entity” approach: a prospective study of 207 patients

2007

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Background: Groin pain remains a major challenge in sports medicine. Aim: To examine 207 consecutive athletes (196 men, 11 women) with groin pain using a standardised and reliable clinical examination programme that focused on signs that suggest pathology in (1) the adductors, (2) the ilopsoas and (3) the rectus abdominis. Patients and methods: Most patients were football players (66%) and runners (18%). In this cohort, the clinical pattern consistent with adductor-related dysfunction, was the primary clinical entity in 58% of the patients and in 69% of the football players. Iliopsoas-related dysfunction was the primary clinical entity in 36% of the patients. Rectus abdominis-related dysfunction was found in 20 (10%) patients but it was associated with adductor-related pain in 18 of these patients. Multiple clinical entities were found in 69 (33%) patients; of these, 16 patients had three clinical entities. Conclusions: These descriptive data extend previous findings that physical examination for groin pain can be reliable. While underscoring the prevalence of adductor-related physical examination abnormality in football players, the data highlight the prevalence of examination findings localising to the iliopsoas among this cohort. Also, the fact that combinations of clinical entities were present has important implications for treatment. The finding of multiple abnormal clinical entities also raises the possibility that earlier presentation may be prudent; it is tempting to speculate that one clinical entity likely precedes other developing entities. These data argue for the need for a trial where clinical entities are correlated with systematic investigation including MRI and ultrasonography.

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“…Various authors [ 36 , 41 , 42 ] have pointed out the frequency of pre-TGA situations involving a Valsalva-like maneuver. A summary [ 14 ] of 12 TGA studies involving 837 patients provided further evidence of a preceding Valsalva-like maneuver (in descending frequency: physical activity, sexual intercourse, swimming) in 44% (26–85%) of TGA patients.…”

Section: Pathophysiologymentioning

confidence: 99%

Guideline “Transient Global Amnesia (TGA)” of the German Society of Neurology (Deutsche Gesellschaft für Neurologie): S1-guideline

Sander¹,

Bartsch²,

Connolly³

et al. 2023

Neurol. Res. Pract.

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Introduction In 2022 the DGN (Deutsche Gesellschaft für Neurologie) published an updated Transient Global Amnesia (TGA) guideline. TGA is characterized by a sudden onset of retrograde and anterograde amnesia for a period of one to a maximum of 24 h (with an average of 6 to 8 h). The incidence is estimated between 3 and 8 per 100,000 population/year. TGA is a disorder that occurs predominantly between 50 and 70 years. Recommendations The diagnosis of TGA should be made clinically. In case of an atypical clinical presentation or suspicion of a possible differential diagnosis, further diagnostics should be performed immediately. The detection of typical unilateral or bilateral punctate DWI/T2 lesions in the hippocampus (especially the CA1 region) in a proportion of patients proves TGA. The sensitivity of MRI is considered higher when performed between 24 and 72 h after onset. If additional DWI changes occur outside the hippocampus, a vascular etiology should be considered, and prompt sonographic and cardiac diagnostics should be performed EEG may help to differentiate TGA from rare amnestic epileptic attacks, especially in recurrent amnestic attacks. TGA in patients < 50 years of age is a rarity, therefore it is mandatory to rapidly search for other causes in particular in younger patients. The cause of TGA is still unknown. Numerous findings in recent years point to a multifactorial genesis. Because the pathomechanism of TGA is not yet clearly known, no evidence-based therapeutic or prophylactic recommendations can be made. Conclusions There is no evidence for chronic sequelae of TGA with respect to cerebral ischemia, chronic memory impairment, or the onset of dementia-related syndromes.

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Das Ilioinguinalis-Syndrom^*

Cited by 17 publications

References 0 publications

Tarsal tunnel syndrome: Electrophysiological study

Tarsal tunnel syndrome: Electrophysiological study

Long-standing groin pain in sportspeople falls into three primary patterns, a “clinical entity” approach: a prospective study of 207 patients

Guideline “Transient Global Amnesia (TGA)” of the German Society of Neurology (Deutsche Gesellschaft für Neurologie): S1-guideline

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Das Ilioinguinalis-Syndrom*

Cited by 17 publications

References 0 publications

Tarsal tunnel syndrome: Electrophysiological study

Tarsal tunnel syndrome: Electrophysiological study

Long-standing groin pain in sportspeople falls into three primary patterns, a “clinical entity” approach: a prospective study of 207 patients

Guideline “Transient Global Amnesia (TGA)” of the German Society of Neurology (Deutsche Gesellschaft für Neurologie): S1-guideline

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Product

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About

Das Ilioinguinalis-Syndrom^*