Cystoid macular oedema after complicated cataract surgery and implantation of an anterior chamber lens

Abstract

During the period January 1988 to August 1990, 48 anterior chamber intraocular lenses were implanted during primary cataract surgery. In 6 eyes an anterior chamber intraocular lens was implanted after intracapsular cataract extraction, in 42 eyes after extracapsular cataract extraction complicated by posterior capsular or zonular rupture. Of the 40 cases which had been followed for at least 6 months (mean 23 months), 5 eyes (12.5%) developed clinically significant cystoid macular oedema. All these eyes had an extracapsular cataract extraction with a posterior capsular tear and anterior vitrectomy and in addition all had some other surgical or postoperative complications. Thus, in this series of complicated cataract surgery, cystoid macular oedema was an important cause of visual impairment but cystoid macular oedema was hardly due to the presence of an anterior chamber intraocular lens.

Pseudophakic Cystoid Macular Edema

“…Vitreous loss increases the prevalence of CME by 10–20% [7,25,26]. Different studies have confirmed that the rate of clinical CME associated with vitreous loss is higher compared to cases without this complication after either extracapsular cataract extraction or phacoemulsification [25,27,28,29]. The lower rate of CME occurring when there is vitreous loss during phacoemulsification has also been explained by the small wound construction and greater stability in this type of surgery compared with large-incision cataract surgery [17,30,31,32].…”

A case of bilateral cystoid macular edema (Irvine—Gass) after bilateral multifocal intraocular lens implantation

“…Около 20% пациентов после катарактальной хирур-гии, по данным ФАГ, страдают КМО различной сте-пени [13][14]. Тем не менее снижение остроты зре-ния клинически определяют только в 1% случаев [15]. При таких осложнениях факохирургии, как разрыв задней капсулы, потеря стекловидного тела, травма радужки, ущемление стекловидного тела в ране, имплантация переднекамерной интраокуляр-ной линзы (ИОЛ), достоверно повышается (до 20%) частота КМО [16].…”

Intravitreal dexamethasone implant for recurrent cystoid macular edema due to Irvine–Gass syndrome: a prospective case series

“…The most accepted patho-physiologic mechanism for PCME is increased inflammation, [1][2][3][4][5] although vitreous disturbance 6 and hypotony 1,2 may account for a sizeable proportion of cases. It typically presents 4-12 weeks after cataract surgery, and often resolves spontaneously.…”

“…4 PCME or Irvine-Gass syndrome develops independently of comorbidities such as diabetes mellitus, vein occlusion, or uveitis. [1][2][3][4][5] Prophylaxis is suggested for in situations where patients who are prone to develop cystoid macular edema following cataract surgery, such as patients with uveitic cataract 7 and those with diabetes. 8,9 In cases that do not resolve spontaneously, a course of topical steroids and non-steroidal anti-inflammatory agents (NSAIDS) helps in the resolution of a large proportion of cases.…”

“…Pseudophakic cystoid macular edema due to Irvine-Gass syndrome [1][2][3][4][5] remains an important cause of decreased vision following uncomplicated cataract surgery and intraocular lens (IOL) implantation. The most accepted patho-physiologic mechanism for PCME is increased inflammation, [1][2][3][4][5] although vitreous disturbance 6 and hypotony 1,2 may account for a sizeable proportion of cases.…”