1963
DOI: 10.1159/000254904
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Cutis Punctata Linearis Colli: Stippled Skin

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Cited by 19 publications
(3 citation statements)
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“…We conclude that this familiar disorder is a new autonomous clinical syndrome with the following main characteristics: its appearance during puberty or just afterwards, the monstrous nature of the sebaceous hyperplasia, the selective localization on the face, the neck and upper thorax, the lack of involvement of peri-orificial regions, the absence of acneiform lesions, the slow progressive nature of the disorder ultimately leading to an extremely unpleasant appearance, the ineffectiveness of conventional acne treatments, and histopathologically, a voluminous sebaceous hyperplasia with lack of inflammation. This rather unique disorder merits discussion regarding its relationship to certain other sebaceous gland disorders: (i) a common seborrhoea oleosa: our patients clearly presented with a considerable seborrhoea, but the most important sign was the sebaceous hyperplasia, entirely abnormal, even for an extreme seborrhoeai (2) an acne or an acneiform eruption: our patients exhibited no clinical or histological signs of the slightest inflammatory acneiform process; (3) a cutis punctata linearis colli (Even-Paz & Sagher, 1963): this disorder appears later in life, spares the face and corresponds to a cutaneous atrophy and not to a sebaceous hyperplasia; it is often observed in cases of solar atrophy or senile atrophy; (4) familial comedones: this particular and relatively rare disorder recently reported by Dantu et al (1978) represents an important differential diagnosis in relation to the cases we report here. The syndrome of familial comedomes appears at about 10 years of age and is characterized by comedones widely spread over the whole body with crateriform depressions containing a comedo which rapidly reforms if removed.…”
Section: Discussionmentioning
confidence: 77%
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“…We conclude that this familiar disorder is a new autonomous clinical syndrome with the following main characteristics: its appearance during puberty or just afterwards, the monstrous nature of the sebaceous hyperplasia, the selective localization on the face, the neck and upper thorax, the lack of involvement of peri-orificial regions, the absence of acneiform lesions, the slow progressive nature of the disorder ultimately leading to an extremely unpleasant appearance, the ineffectiveness of conventional acne treatments, and histopathologically, a voluminous sebaceous hyperplasia with lack of inflammation. This rather unique disorder merits discussion regarding its relationship to certain other sebaceous gland disorders: (i) a common seborrhoea oleosa: our patients clearly presented with a considerable seborrhoea, but the most important sign was the sebaceous hyperplasia, entirely abnormal, even for an extreme seborrhoeai (2) an acne or an acneiform eruption: our patients exhibited no clinical or histological signs of the slightest inflammatory acneiform process; (3) a cutis punctata linearis colli (Even-Paz & Sagher, 1963): this disorder appears later in life, spares the face and corresponds to a cutaneous atrophy and not to a sebaceous hyperplasia; it is often observed in cases of solar atrophy or senile atrophy; (4) familial comedones: this particular and relatively rare disorder recently reported by Dantu et al (1978) represents an important differential diagnosis in relation to the cases we report here. The syndrome of familial comedomes appears at about 10 years of age and is characterized by comedones widely spread over the whole body with crateriform depressions containing a comedo which rapidly reforms if removed.…”
Section: Discussionmentioning
confidence: 77%
“…these regions the papules were not confluent and did not form a patch. The individual lesions were not randomly dispersed, but rather were linear, as in cutis punctata linearis colli (Even-Paz & Sagher, 1963). Their distribution represents the hypertrophy of the normal sebaceous glands ( Fig.…”
mentioning
confidence: 99%
“…Histology was referable to sebaceous gland hyperplasia, which in their case series was found to be induced by corticosteroid use. 3 Some authors conclude that the two conditions are variants of the same entity, with slight differences in the degree of sebaceous hyperplasia, 4,5 despite a certain later work reporting small clinical differences between the two. 6 Research into the reasons for the linear arrangements of lesions produced two schools of thought.…”
Section: Methodsmentioning
confidence: 98%