“…An evaluation of the techniques currently in use shows that the conventional technique of infraclavicular subclavian vein catheterization eliminates the neurovascular complications encountered in catheterizing the internal jugular vein, though it carries the risk of subclavian artery puncture and a high risk of significant late stenosis (Borja, 1972). The latter complication is thought to result from the pressure of the catheter on the caudal aspect of the subclavian vein as it bends to enter the superior vena cava (Barrett et al, 1988;Schillinger et al, 1991).…”
Section: Discussionmentioning
confidence: 99%
“…Complication rates range between 0.1-10% (Seneff, 1991). Acute complications, with an incidence of 7.2%, are incurred during catheter insertion and are basically iatrogenic (Davidson et al, 1963;Yarom, 1964;Yoffa, 1965;Borja, 1972;Ryan et al, 1974;Voegele, 1976;Herbst, 1978;Goutail-Flaud et al, 1991;Yerdel et al, 1991a,b;Ballard et al, 1992).…”
Central venous access is important in both assessment and treatment of the patient. In modern clinical practice, a percutaneous approach is preferred. The well-established subclavian and internal jugular vein catheterization techniques, however, still carry the risk of major acute complications. In this article we describe a trial study of a percutaneous brachiocephalic vein catheterization technique conducted on a total of 74 cadavers undergoing autopsy. Relying on constant and easily recognizable anatomical landmarks, we performed a total of 128 catheterizations of the right and left brachiocephalic veins. The success of catheterization was ascertained by regional dissection. Our results show very high reproducibility with a success rate of about 97% on the first attempt of catheterization on both right and left sides. Equally important, collateral injury of neighboring structures was consistently absent. We propose the introduction of this rather overlooked approach into clinical practice.
“…An evaluation of the techniques currently in use shows that the conventional technique of infraclavicular subclavian vein catheterization eliminates the neurovascular complications encountered in catheterizing the internal jugular vein, though it carries the risk of subclavian artery puncture and a high risk of significant late stenosis (Borja, 1972). The latter complication is thought to result from the pressure of the catheter on the caudal aspect of the subclavian vein as it bends to enter the superior vena cava (Barrett et al, 1988;Schillinger et al, 1991).…”
Section: Discussionmentioning
confidence: 99%
“…Complication rates range between 0.1-10% (Seneff, 1991). Acute complications, with an incidence of 7.2%, are incurred during catheter insertion and are basically iatrogenic (Davidson et al, 1963;Yarom, 1964;Yoffa, 1965;Borja, 1972;Ryan et al, 1974;Voegele, 1976;Herbst, 1978;Goutail-Flaud et al, 1991;Yerdel et al, 1991a,b;Ballard et al, 1992).…”
Central venous access is important in both assessment and treatment of the patient. In modern clinical practice, a percutaneous approach is preferred. The well-established subclavian and internal jugular vein catheterization techniques, however, still carry the risk of major acute complications. In this article we describe a trial study of a percutaneous brachiocephalic vein catheterization technique conducted on a total of 74 cadavers undergoing autopsy. Relying on constant and easily recognizable anatomical landmarks, we performed a total of 128 catheterizations of the right and left brachiocephalic veins. The success of catheterization was ascertained by regional dissection. Our results show very high reproducibility with a success rate of about 97% on the first attempt of catheterization on both right and left sides. Equally important, collateral injury of neighboring structures was consistently absent. We propose the introduction of this rather overlooked approach into clinical practice.
“…In 1979, the peelaway sheath was introduced, and this simplified the percutaneous subclavian placement of pacemaker leads [3]. Although there were substantial risks related to subclavian vein cannulation [4], this became the most popular method for implantation of permanent pacemakers [5], because it was speedy and relatively atraumatic, and suitable for the placement of multiple leads.…”
“…Finally, frequent use of central venous catheters has resulted in an increased documentation of air embolism through intravenous equipment over the last 30 years. [7][8][9][10] Therapeutic and diagnostic automated intravenous infusion devices are probably responsible for a small percentage of VAE through intravenous catheters, with most occurring during placement, use, and removal of central venous lines. Table 26.1 lists the ever-broadening clinical settings in which VGE has been reported.…”
Section: Venous Gas Embolism Historical Perspective and Etiologymentioning
DefinitionsGas embolism refers to the abnormal presence of gas within the circulatory system. It is a known complication of various surgical, therapeutic, and diagnostic procedures. It can also occur as a result of trauma. Gas embolism may be asymptomatic or may even result in immediate cardiovascular collapse. The type and severity of sequelae depend on the composition and amount of gas, the rate and location of entry, and on patient characteristics such as size, cardiopulmonary reserve, and presence of an intracardiac rightleft communication. An understanding of the etiologies and pathophysiology of gas embolism is important in order to recognize, treat, and, most importantly, prevent this potentially catastrophic complication.Gas enters the circulation when two conditions co-exist: (1) a communication between a gas source and the vascular system, and (2) a pressure gradient that favors gas entry into the vasculature. When the atmosphere is the source of gas, then this condition is termed air embolism. Gas can be entrained into open veins or venous sinuses, insufflated into traumatized vessels, or accidentally infused under pressure through indwelling venous or arterial catheters. Rarely, dissolved gas is released in blood through effervescence caused by rapid re-warming or decompression. 1 Venous (pulmonary) gas embolism (VGE) refers to gas that gains entry into the venous system and causes morbidity via its effects on the right heart and pulmonary vasculature. Systemic (arterial) gas embolism (SGE) results from gas that is propelled by the left heart and embolizes to systemic arterial beds causing ischemia and infarction. Paradoxical gas embolism (PGE) originates in the venous system and gains access to the arterial system through intracardiac defects or the pulmonary circulation.
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