Crescentic Glomerulonephritis Complicating Idiopathic Membranous Glomerulonephropathy

Ja, Mitas; Lr, Frank; Ar, Swerdlin; Dl, Johnson; Gm, Rabetoy

doi:10.1097/00007611-198305000-00036

Cited by 9 publications

(2 citation statements)

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“…Additional case reports which failed to satisfy these crite ria have been excluded [2,9,[16][17][18][19]. There are 4 pub lished cases (table I), other than our own, of well docu mented preexisting MN evolving into AC-RPGN.…”

Section: Discussionmentioning

confidence: 99%

Progression of Membranous Nephropathy to Acute Crescentic Rapidly Progressive Glomerulonephritis and Response to Pulse Methylprednisolone

Jd¹,

Js²,

Jl³

et al. 1986

Am J Nephrol

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There have been only sporadic reports of membranous nephropathy (MN) evolving into acute crescentic rapidly progressive glomerulonephritis (AC-RPGN). A patient with MN developed acute oliguric renal failure with a serum creatinine (SCr) of 8.4 mg/dl after 5 years of normal renal function. Biopsy now revealed epithelial crescent formation superimposed on MN. Pulse methylprednisolone resulted in significant improvement in renal function, with a SCr of 2.2 mg/dl at 6 months. No other favorable outcomes occurred in the 4 previous case reports of MN evolving into RPGN. AC-RPGN should be considered a treatable etiology of acute renal failure in the setting of MN.

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Section: Discussionmentioning

confidence: 99%

Progression of Membranous Nephropathy to Acute Crescentic Rapidly Progressive Glomerulonephritis and Response to Pulse Methylprednisolone

Jd¹,

Js²,

Jl³

et al. 1986

Am J Nephrol

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“…All 6 patients with anti-GM antibodies developed renal failure. Of the remaining 7 patients, all were anti-GBM antibody-negative, 1 recovered renal function without therapy [11], 1 developed end-stage kidney disease despite steroid therapy [1], 3 responded to steroid therapy and had stable chronic renal insufficiency [15,18,19] and 2 patients had no available follow-up information [12,16]. Various forms of therapy have been used including high-dose steroids, azathioprine, cyclophosphamide, antiplatelet agents and plasmapheresis.…”

Section: Discussionmentioning

confidence: 99%

Recurrent Crescentic Membranous Nephropathy in Two Successive Renal Transplants: Association with Choroidal Effusions and Retinal Detachment

Lazowski

Sablay

Glicklich³

1998

Am J Nephrol

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We report the case of a 50-year-old man in whom crescentic membranous nephropathy recurred in two succesive renal transplants leading rapidly to renal failure. Deterioration of renal function was associated with choroidal effusions and retinal detachments. Multiple serologic tests were negative. High-dose steroids, cyclophosphamide, cyclosporine, plasmapheresis and OKT3 failed to arrest the deterioration of renal function.

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Immune and Inflammatory Glomerular Diseases

Manoharon

Schelling

Diamond

et al. 2013

Seldin and Giebisch's the Kidney

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Crescentic Glomerulonephritis Complicating Idiopathic Membranous Glomerulonephropathy

Cited by 9 publications

References 0 publications

Progression of Membranous Nephropathy to Acute Crescentic Rapidly Progressive Glomerulonephritis and Response to Pulse Methylprednisolone

Progression of Membranous Nephropathy to Acute Crescentic Rapidly Progressive Glomerulonephritis and Response to Pulse Methylprednisolone

Recurrent Crescentic Membranous Nephropathy in Two Successive Renal Transplants: Association with Choroidal Effusions and Retinal Detachment

Immune and Inflammatory Glomerular Diseases

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