Cranial CT scans in eating disorder patients and controls

Lankenau, Helen; Swigar, Mary E.; Bhimani, Sultan; Quinlan, Donald M.; Luchins, David

doi:10.1016/0010-440x(85)90034-3

Cited by 42 publications

(30 citation statements)

References 19 publications

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“…Neuroimaging studies with CT reported cerebral atrophy and enlarged ventricles in ill AN (98)(99)(100)(101)(102)(103)(104)(105)(106). In BN similar but less pronounced structural brain abnormalities were reported (107) and may have been related to a chronic dietary restriction.…”

Section: Brain Structurementioning

confidence: 75%

Neurobiology of anorexia and bulimia nervosa

Kaye

2008

Physiology & Behavior

521

369

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Section: Brain Structurementioning

confidence: 75%

Neurobiology of anorexia and bulimia nervosa

Kaye

2008

Physiology & Behavior

521

369

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“…Characteristics of chronicity of anorexia nervosa, such as vomiting, laxative abuse (Garfinkel, Moldofsky, & Garner, 1977;Halmi, Brodland, & Loney, 1973;Hsu et al, 1979;Steinhausen, 1983), and extreme loss of weight (Dally, 1969;Hsu et al, 1979;Morgan & Russel, 1975), are associated with ventricular enlargement (Hoffman et al, 1989;Krieg et al, 1988;Lankenau, Swigar, Bhimani, Luchins, & Quinlan, 1985;Nussbaum, Shenker, Marc, & Klein, 1980). Artmann et al (1985) have shown that anorexic patients with histories of PBCs, especially low birth weight, exhibited a higher rate of brain abnormalities, and were less likely to show improvement in ventricular size on refeeding, than those without PBCs.…”

Section: Chronicity Of Anorexia Nervosa: Neurological Abnormalities Amentioning

confidence: 99%

Chronicity in anorexia nervosa: Pregnancy and birth complications as risk factors

Bakan

Birmingham

Goldner

1991

Int. J. Eat. Disord.

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The chronic form of anorexia nervosa (AN) affects about one‐quarter of patients and has serious somatic and psychological consequences. This paper presents evidence in support of the hypothesis that pregnancy and birth complications (PBCs) associated with neuropathology may be risk factors for chronic anorexia nervosa. There is increasing evidence that PBCs are risk factors for other psychiatric and somatic disorders. Recent findings of possible premorbid neuropathology in patients with anorexia nervosa suggest that PBCs may be risk factors for chronicity in anorexia nervosa. Topics discussed include the origins of constitutional vulnerability to anorexia nervosa, correlates of PBCs and of chronic anorexia nervosa, the relationship of PBCs to neurological and neurochemical abnormalities, chronicity of anorexia nervosa in relation to PBCs and to neurological and neurochemical abnormalities, and the research implications of the hypothesis that PBCs and associated neuropathology are risk factors for chronicity in anorexia nervosa.

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“…However, recent work has focused on the identification of a central nervous system dysfunction in eating disorders and has demonstrated abnormal findings in computed tomographic brain scans [1,2], positron emission tomography [3], neurotransmitter [4,5], and neuropeptide levels [6], and brain event-related potentials (ERPs) [7].…”

Section: Introductionmentioning

confidence: 99%

Prolonged P300 Latency in Eating Disorders

Otagaki

Tohoda²,

Osada³

et al. 1997

Neuropsychobiology

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This study investigated event-related potential (ERP) indices of information-processing in eating disorders. ERPs during an auditory two-tone discrimination task were recorded at midline at 3 sites in 28 anorexic patients, 12 low-weight bulimic patients (body mass index (BMI) under 17.5), 12 normal-weight bulimic patients (BMI over 17.5), and 40 control subjects. The P300 latency was significantly prolonged at all sites in both bulimic groups compared with that in controls, and at frontal and central electrode sites in anorexics. In contrast, the P300 amplitude did not differ between these groups at any site. The prolonged P300 latency in eating disorders suggests a task-related slowing of perceptual decisions that reflects their cognitive impairment.

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Cranial CT scans in eating disorder patients and controls

Cited by 42 publications

References 19 publications

Neurobiology of anorexia and bulimia nervosa

Neurobiology of anorexia and bulimia nervosa

Chronicity in anorexia nervosa: Pregnancy and birth complications as risk factors

Prolonged P300 Latency in Eating Disorders

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