Coxsackie B myopericarditis in adults

Smith, W.G.

doi:10.1016/0002-8703(70)90035-9

Cited by 189 publications

(70 citation statements)

References 25 publications

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“…5,6) These ECG abnormalities do not always resolve completely and persist into the late phase. [7][8][9] In a study by Smith 8) that followed 22 patients with Coxsackie B myocarditis for 6 months to 6 years, 6 patients had persistent ECG abnormalities. We also have described a patient with myocarditis with advanced AV block and severe cardiac failure during the acute phase, with the AV block persisting in the late phase but no wall motion abnormalities.…”

Section: Discussionmentioning

confidence: 99%

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Endomyocardial Biopsy Findings in 50 Patients with Idiopathic Atrioventricular Block. Presence of myocarditis.

Morimoto

Hiramitsu

2001

Jpn Heart J

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Section: Discussionmentioning

confidence: 99%

“…[7][8][9] However, despite the fact that AV block develops in some patients with myocardial disease, few studies 10) have been performed to detect myocardial lesions in patients with conduction disturbances. The present study was therefore performed to analyze myocardial lesions histologically in a large series of patients with AV conduction disturbances.…”

mentioning

confidence: 99%

Endomyocardial Biopsy Findings in 50 Patients with Idiopathic Atrioventricular Block. Presence of myocarditis.

Morimoto

Hiramitsu

2001

Jpn Heart J

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“…It is nowbelieved that most cases of acute myocarditis are induced by viral infection (1)(2)(3). Electrocardiographic findings of these cases often mimic AMI (4)(5)(6)(7).…”

Section: Introductionmentioning

confidence: 99%

Serial Electrocardiographic Findings in Acute Myocarditis.

1994

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The purpose of the present study was to clarify the characteristic findings of electrocardiogram (ECG) in ll patients with acute myocarditis. ST elevation without reciprocal ST depression was one of the conspicuous findings in the acute stage. Total QRSamplitudes at the acute stage were significantly decreased as compared to those before illness and during the convalescent stage. Abnormal Q waves were present in 7 patients and disappeared in a short period. The number of leads showing Q waves was inversely correlated to left ventricular (LV) ejection fraction (r=-0.87, p<0.01). Conduction disturbances were present in 7 patients. Second degree and advanced AV block was transient while bundle branch block remained over months. Corticosteroid treatment was effective for patients whohad edematous myocardial thickening and AVconduction disturbances. As the serial ECGfindings in acute myocarditis are so characteristic, and this help to differentiate it from acute myocardial infarction (AMI). (Internal Medicine 33: 659-666, 1994)

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“…Yet, cases of persistent symptoms and cardiac manifestations with or without eventual recovery have been well documented (2,3). Although chronic myocarditis and myocardial fibrosis have been postulated as sequelae of acute viral myocarditis, the evidence in man remains circumstantial (4).…”

Section: Introductionmentioning

confidence: 99%

Heart muscle performance after experimental viral myocarditis.

Adesanya¹,

Goldberg²,

Phear³

et al. 1976

J. Clin. Invest.

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A B S T R A C T As part of an inquiry into possible antecedents of idiopathic cardiomyopathy, acute experimental coxsackie virus myocarditis was studied for late structural and functional sequelae. Myocarditis was induced in 12-and 22-day-old hamsters by inoculation with coxsackie virus B3. Early viremia occurred, followed by virus replication in heart muscle.Maximum peak developed tension (Tpd) of isometrically contracting isolated heart muscle was depressed 17 and 43% in the animals inoculated at 12 days, and studied 18 and 90 days later, respectively, as compared to their uninoculated controls. In both infected groups, less muscle stretch was required to reach the length at which Tpd was produced. Animals studied 180 days after inoculation did not differ from controls.The muscles from animals inoculated at 22 days of age and studied 18 days later showed a 15% depression of Tpd compared to their controls. Glycerinated muscles from this infected group developed 50% less tension than their controls. The muscles of hamsters inoculated with virus at 22 days and studied 90 and 180 days later showed no change in Tpd.The data suggest that contractility and compliance of heart muscle are decreased 18 days after inoculation, but recover by 90 days if the animals are inoculated at age 22 days. However, if the animals are inoculated at a younger age (12 days), depression of myocardial performance persists for at least an additional 90 days.It is concluded that the inflammatory stage of experimental acute coxsackie virus B3 myocarditis in the

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Coxsackie B myopericarditis in adults

Cited by 189 publications

References 25 publications

Endomyocardial Biopsy Findings in 50 Patients with Idiopathic Atrioventricular Block. Presence of myocarditis.

Endomyocardial Biopsy Findings in 50 Patients with Idiopathic Atrioventricular Block. Presence of myocarditis.

Serial Electrocardiographic Findings in Acute Myocarditis.

Heart muscle performance after experimental viral myocarditis.

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