Correction to: Assessing immuno‑expression of p53 protein and TP 53 gene amplification in histologically negative surgical margins of oral squamous cell carcinoma patients and normal oral mucosa

Kamat, Mamata; Puranik, Rudrayya S; Das, A Bhagavan; Patil, B R; Patil, Shankargouda

doi:10.1007/s00784-022-04834-x

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“…The p53 mutation has been noticed in over 50% of human cancers. 18 Kamat et al 19 suggested that p53 expression status in oral carcinoma can be used to identify the chances of developing local recurrence as well as to predict survival outcomes. Novack et al 20 reported that p53 expression was abnormal in oral epithelial dysplasia.…”

Section: Resultsmentioning

confidence: 99%

Ursolic Acid-Loaded Chitosan Nanoparticles Modulate the Expression Pattern of Apoptotic Markers Towards Oral Tumour Inhibition in Golden Syrian Hamsters

2023

TJNPR

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Oral cancer is an aggressive life-threatening cancer that causes a severe health burden and high mortality around the globe. Oral cancer affects approximately 0.4 million people every year globally. Tobacco and alcohol abuse are pointed out as potent oral carcinoma risk factors. 1 Oral carcinoma frequently arises on lips, gums, tongue, palates and salivary glands. The treatment of oral carcinoma still faces significant difficulties because of its rapid progression, resistance to drugs, and lack of response, despite advancements in oral cancer treatments such as targeted therapy, gene therapy, and therapy with potent chemotherapeutic drugs. Early detection, prevention and early treatment can significantly improve oral cancer patients' life quality. 2,3 Cells with potentially harmful mutations are eliminated by apoptosis, an ordered and coordinated sequential process. Cancer cells avoid apoptosis by interfering with the actions of pro-apoptotic and antiapoptotic proteins. A defect in the apoptotic mechanism would result in uncontrollable and abnormal growth of cells that ultimately leads to the formation of a tumor. Cellular apoptosis has been aberrantly dysregulated at various stages of oral squamous cell carcinoma. 4,5

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Section: Resultsmentioning

confidence: 99%

Ursolic Acid-Loaded Chitosan Nanoparticles Modulate the Expression Pattern of Apoptotic Markers Towards Oral Tumour Inhibition in Golden Syrian Hamsters

2023

TJNPR

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Correction to: Assessing immuno‑expression of p53 protein and TP 53 gene amplification in histologically negative surgical margins of oral squamous cell carcinoma patients and normal oral mucosa

Cited by 1 publication

References 0 publications

Ursolic Acid-Loaded Chitosan Nanoparticles Modulate the Expression Pattern of Apoptotic Markers Towards Oral Tumour Inhibition in Golden Syrian Hamsters

Ursolic Acid-Loaded Chitosan Nanoparticles Modulate the Expression Pattern of Apoptotic Markers Towards Oral Tumour Inhibition in Golden Syrian Hamsters

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