Coronary Sclerosis, Myocardial Infarction, Myocarditis, Cardiomyopathy, Coronary Anomalies, and the Cardiac Conduction System

Dettmeyer, R.

doi:10.1007/978-3-642-20659-7_13

Cited by 4 publications

(2 citation statements)

References 282 publications

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“…Even though there are numerous articles (often case reports) identifying as an underlying cause of death, the overall risk increase for sudden death generated by this pathology is disputed . It was associated with various potentially lethal cardiovascular pathologies, including ventricular rupture , life‐threatening arrhythmias , acute myocardial infarction (AMI) , hypertrophic cardiomyopathy , or apical ballooning syndrome .…”

Section: Discussionmentioning

confidence: 99%

Myocardial Bridging: A Meta‐Analysis of Prevalence

Hostiuc

Negoi

Rusu

et al. 2017

Journal of Forensic Sciences

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The main objective of this article was to analyze prevalence data about myocardial bridging (MB) in published studies. To this purpose, we performed a meta-analysis of studies published in English literature that contained data about the prevalence of MB and its anatomical characteristics. The overall prevalence was 19% (CI: 17-21%); autopsy studies revealed an overall prevalence of 42% (CI: 30-55%), CT studies 22% (CI: 18-25%), and coronary angiography 6% (CI: 5-8%). Most bridges were located on the left anterior descending artery (82% overall, 63% on autopsy studies), had a mean thickness of 2.47 mm and a mean length of 19.3 mm. In conclusion, autopsy studies should be the gold standard in evaluating the actual prevalence of myocardial bridges, while in vivo high-resolution CT scanning should be preferred to coronary angiography studies.

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Section: Discussionmentioning

confidence: 99%

Myocardial Bridging: A Meta‐Analysis of Prevalence

Hostiuc

Negoi

Rusu

et al. 2017

Journal of Forensic Sciences

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“…Caution should also be taken with the interpretation of presence of CBN, since they can also be observed after CPR including defibrillation and other trauma. Not specifically related to PCI, but clearly of importance in a forensic setting, CBN has been observed in situations of catecholamine excess, drowning, drug abuse, cobalt poisoning, starvation, free radical injuries, brain death and intracerebral haemorrhage [59]. CBN is accompanied by microvascular injury leading to endothelial swelling, microvascular obstruction and interstitial haemorrhages (Fig.…”

Section: Ischemia and Reperfusion And Other Forms Of Injurymentioning

confidence: 99%

Diagnosis of myocardial infarction at autopsy: AECVP reappraisal in the light of the current clinical classification

et al. 2019

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Ischemic heart disease is one of the leading causes of morbidity and death worldwide. Consequently, myocardial infarctions are often encountered in clinical and forensic autopsies, and diagnosis can be challenging, especially in the absence of an acute coronary occlusion. Precise histopathological identification and timing of myocardial infarction in humans often remains uncertain while it can be of crucial importance, especially in a forensic setting when third person involvement or medical responsibilities are in question. A proper post-mortem diagnosis requires not only up-to-date knowledge of the ischemic coronary and myocardial pathology, but also a correct interpretation of such findings in relation to the clinical scenario of the deceased. For these reasons, it is important for pathologists to be familiar with the different clinically defined types of myocardial infarction and to discriminate myocardial infarction from other forms of myocardial injury. This article reviews present knowledge and postmortem diagnostic methods, including post-mortem imaging, to reveal the different types of myocardial injury and the clinicalpathological correlations with currently defined types of myocardial infarction.

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Different degrees of ischaemic injury in the right and left ventricle in cases of severe, nonfatal, pulmonary embolism

et al. 2014

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Pulmonary fat embolism (PFE) is a common complication of blunt force traumas with bone fractures. Severe forms cause impedance to right ventricular (RV) ejection, with eventual right heart ischaemia and failure. In a prospective study, we have investigated 220 consecutive autopsy cases (73 females, 147 males, mean age 52.1 years, min 14 years, max 91 years). PFE was detected in 52 cases that were divided into three groups according to the degree of PFE (1-3). A fourth group of cases of violent death without PFE was used for comparison. In each case, histology (H&E, Masson) and immunohistochemistry (fibronectin and C5b-9) were performed on six cardiac samples (anterior, lateral and posterior wall of both ventricles). The degree of cardiac damage was registered in each sample and the mean degree of damage was calculated in each case at the RV and left ventricle (LV). Moreover, a parameter ∆ that is the difference between the mean damage at the RV and the LV was calculated in each case. The results were compared within each group and between the groups. In the present study, we could not detect prevalent RV damage in cases of high degree PFE as we did in our previous investigation. In the group PFE3 the difference of the degree of damage between the RV and LV was higher than the one observed in the groups PFE0-2 with the antibody anti-fibronectin. Prevalent right ventricular stress in cases of severe PFE may explain this observation.

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Coronary Sclerosis, Myocardial Infarction, Myocarditis, Cardiomyopathy, Coronary Anomalies, and the Cardiac Conduction System

Cited by 4 publications

References 282 publications

Myocardial Bridging: A Meta‐Analysis of Prevalence

Myocardial Bridging: A Meta‐Analysis of Prevalence

Diagnosis of myocardial infarction at autopsy: AECVP reappraisal in the light of the current clinical classification

Different degrees of ischaemic injury in the right and left ventricle in cases of severe, nonfatal, pulmonary embolism

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