, 1976;Wartman and Hellerstein, 1948). This report describes a patient with isolated right ventricular infarction with unusual electrophysiological findings. Her initial electrocardiogram showed atrial escape rhythm with incomplete right bundle-branch block and left posterior hemiblock. Later, she developed atrioventricular (AV) block with supra-and infra-Hisian, "phase 4," conduction defects. The sinus malfunction and high degree AV block persisted over 2 weeks and an atrioventricular sequential pacemaker was implanted. Hymodynamic study showed that her cardiac output was highly dependent on the heart rate and properly timed AV interval, and the pacemaker was programmed accordingly. Case Report A 69-year-old female, without a previous history of heart disease was admitted because of sudden transient loss of consciousness. On physical examination, the blood pressure was 120/70 mmHg, the lungs were clear, and the neck veins distended. There were no abnormal heart sounds nor murmurs. The serum creatine kinase was 200 units with MB fraction of 15 % . Her initial electrocardiogram (Fig. 1) demonstrated sinus arrest with an atrial escape rhythm of 54 beatdmin, a QRS pattern of right bundle-branch block with left posterior hemiblock (RBBB+LPHB) and a QR pattern in V,, mild ST elevation in V,-4 and absence of Q wave in either V2.4 or aVL and L, or in the diaphragmatic leads. These findings are compatible with isolated right ventricular infarction. Within a few minutes, complete AV block developed with a ventricular escape rhythm of 40 beatdmin. Chest radiography showed mild cardiomegaly with clear lung fields. Echocardiography showed that the right ventricle was dilated and contracted poorly. There was no pericardial effusion. Similar findings were obtained using a technetium99m (99mTc) red cell labelled, multiple gated blood pool scan (MUGA) (Fig. 2). A Swan Ganz catheter was inserted and the following pressures were measured: right atrium, 10 mmHg with a "dip and plateau" pattern; pulmonary artery, 34/7 mmHg; wedge pressure, 7 mmHg. The cardiac index was 2.1 I/min per mz. These imaging and hemodynamic findings confirmed the intial diagnosis of acute right ventricular infarction. Isotonic fluids, plasma, and dextran were given intravenously. A temporary atrioventricular pacemaker was inserted transvenously and her condition improved. Seven days later, her atrioventricular conduction improved and 1 : 1 AV response to atrial pacing was observed. However, during repeated attempts to lower the atrial-paced rhythm below 50 beatdmin, complete AV block appeared. Gradual disappearance of the block followed an increase in the atrial pacing rate above 50 beatdmin. The RBBB + LPHB persisted.His bundle recording during high right atrial pacing (Table I) showed marked prolongation of the PA interval, the HV interval was also prolonged and the AH interval was at the upper limit of normal (Puech, 1975). The minimal