Abstract:These findings support the hypothesis that progressive fibrosis at the FHL myotendinous junction can cause increased loading of the hallux MTPJ, thereby leading to hallux rigidus.
“…The key difference in function between intrinsic and extrinsic supinators of the foot is their action around the midtarsal, subtalar and ankle joints, specifically the latter. Perhaps in pes planus, the different posture of the foot disadvantages FHL and FDL so that they need to generate greater forces to contribute the required moments and thus facilitate normal sagittal plane ankle function This preference for FHL and FDL contributions to MLA support may result in hypertrophy [15,18].…”
Section: Discussionmentioning
confidence: 99%
“…Tibialis anterior contracts in early stance to allow gradual plantarflexion of the foot and to decelerate downward motion of the foot. FHL and FDL further contribute to the maintenance of the MLA [16] but their actions are perhaps more coupled with intrinsic muscle and plantar fascia function than TP and TA, since all these structures insert into the digits [17,18]. However, the relationship between the extrinsic and intrinsic foot structures that share common functions has not been reported in pes planus.…”
Greater CSA and thickness of the extrinsic muscles might reflect compensatory activity to support the MLA if the intrinsic foot muscle function has been compromised by altered foot structure. A thinner plantar fascia suggests reduced load bearing, and regional variations in structure and function in feet with pes planus.
“…The key difference in function between intrinsic and extrinsic supinators of the foot is their action around the midtarsal, subtalar and ankle joints, specifically the latter. Perhaps in pes planus, the different posture of the foot disadvantages FHL and FDL so that they need to generate greater forces to contribute the required moments and thus facilitate normal sagittal plane ankle function This preference for FHL and FDL contributions to MLA support may result in hypertrophy [15,18].…”
Section: Discussionmentioning
confidence: 99%
“…Tibialis anterior contracts in early stance to allow gradual plantarflexion of the foot and to decelerate downward motion of the foot. FHL and FDL further contribute to the maintenance of the MLA [16] but their actions are perhaps more coupled with intrinsic muscle and plantar fascia function than TP and TA, since all these structures insert into the digits [17,18]. However, the relationship between the extrinsic and intrinsic foot structures that share common functions has not been reported in pes planus.…”
Greater CSA and thickness of the extrinsic muscles might reflect compensatory activity to support the MLA if the intrinsic foot muscle function has been compromised by altered foot structure. A thinner plantar fascia suggests reduced load bearing, and regional variations in structure and function in feet with pes planus.
“…Periods of power generation suggest that active mechanisms are responsible 27. The first metatarsal plantar flexion ROM, therefore, may be the result of (1) muscle control of the intrinsic muscles,24 (2) flexor hallucis longus muscle,20,21 (3) Achilles tendon forces acting through the plantar fascia,10 and/or (4) some combination of these actions, which are retained in individuals with PTTD.…”
abnormal kinematics during a heel rise task. 41 The normal combined action of the posterior tibialis and triceps surae muscles is thought to produce ankle plantar flexion with inversion during a heel rise task. 15,23,33 Clinically, an
“…Kirane et al investigated the role of stenosing tenosynovitis of the flexor hallucis longus tendon, as manifested by limited distal excursion, in the etiology of hallux rigidus 25 . Cadaver specimens were placed in a Robotic Dynamic Activity Simulator to simulate the stance phase of gait.…”
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