Contralateral Amaurosis After Retrobulbar Injection

Friedberg, Howard L.; Kline, Oram R.

doi:10.1016/0002-9394(86)90770-1

Cited by 75 publications

(13 citation statements)

References 10 publications

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“…Although rare, contralateral transient vision loss after retrobulbar anesthesia has been reported. [2][3][4] To our knowledge, this is the first reported case of contralateral vision loss and oculomotor nerve palsy following peribulbar anesthesia.…”

Section: Discussionmentioning

confidence: 82%

Vision loss and partial third nerve palsy following contralateral peribulbar anesthesia

Krilis

Zeldovich

Garrick

et al. 2013

Journal of Cataract and Refractive Surgery

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We report the case of a 72-year-old woman who experienced transient complete visual loss and a partial third nerve palsy in 1 eye following cataract surgery under local anesthesia in the fellow eye. Symptoms and signs were determined to result from the administration of a peribulbar block, which was presumably associated with ipsilateral transoptic nerve sheath spread. We believe this is the first report of contralateral amaurosis and oculomotor nerve palsy following peribulbar anesthesia.

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Section: Discussionmentioning

confidence: 82%

Vision loss and partial third nerve palsy following contralateral peribulbar anesthesia

Krilis

Zeldovich

Garrick

et al. 2013

Journal of Cataract and Refractive Surgery

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show abstract

“…On the other hand, there was a simultaneous rise in the right nontraumatized nerves during this period. This may be indirect evidence of Friedberg and Kline's [12] proposal that superoxide prod ucts have the potential to migrate to the contralateral optic nerve through the chiasm.…”

Section: Discussionmentioning

confidence: 95%

Ultrastructural Findings and SOD Levels in Experimental Optic Nerve Injury: Effect of Thyroid-Releasing Hormone on the Degenerative and Regenerative Processes

Haciyakupoğlu¹,

Özdemir

Polat

et al. 1994

Ophthalmologica

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We performed pressure on the left optic nerves of 24 pigs and injected intravenous thyroid-releasing hormone (TRH) to 12 of these pigs in order to evaluate the degeneration and regeneration in the optic nerves. The histopathologic, ultra-structural and biochemical examinations of the optic nerves were made at the 24th hour, 7th, 15th and 30th days. Although the light-microscopic examinations were normal, ultrastructural changes of the uncompressed optic nerves were interesting. Histopathologic and ultrastructural investigation of the compressed optic nerves showed significant degenerative changes in the non-TRH-applied group. Ultrastructural comparison yielded lighter degenerative changes in the TRH-applied group but there was no clue showing the stimulation of regeneration. We observed increased superoxide dismutase (SOD) levels in the nontraumatized optic nerves due to the cellular stress. The SOD values were found to be low in highly damaged left compressed optic nerves indicating the prevention of SOD enzyme synthesis.

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“…No 1/3 final da introdução da agulha é conveniente solicitar que olhe para baixo e levemente para fora (2,8,19) . Esta posição afasta o nervo óptico da extremidade da agulha.…”

Section: Discussionunclassified

Oclusão artério-venosa da retina após bloqueio retrobulbar: relato de dois casos

Torres¹,

Luchini²,

Weis³

et al. 2005

Arq. Bras. Oftalmol.

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Contralateral Amaurosis After Retrobulbar Injection

Cited by 75 publications

References 10 publications

Vision loss and partial third nerve palsy following contralateral peribulbar anesthesia

Vision loss and partial third nerve palsy following contralateral peribulbar anesthesia

Ultrastructural Findings and SOD Levels in Experimental Optic Nerve Injury: Effect of Thyroid-Releasing Hormone on the Degenerative and Regenerative Processes

Oclusão artério-venosa da retina após bloqueio retrobulbar: relato de dois casos

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