Conserved Role of the Large Conductance Calcium-Activated Potassium Channel, K<sub>Ca</sub>1.1, in Sinus Node Function and Arrhythmia Risk

Pined, Santiago; Nikolova-Krstevski, Vesna; Leimena, Christiana; Atkinson, Andrew; Altekoester, Ann-Kristin; Cox, Charles D.; Jacoby, Arie S.; Huttner, Inken G.; Ju, Yue-Kun; Soka, Magdalena; Ohanian, Monique; Trived, Gunjan; Kalvakuri, Sreehari; Johnson, Renée; Molenaar, Peter; Kuchar, Dennis L.; Allen, David G.; Helden, Dirk van; Harvey, Richard P.; Hill, Adam P.; Bodmer, Rolf; Vogler, Georg; Dobrzyn, Halina; Ocorr, Karen; Fatkin, Diane

doi:10.1101/2020.06.28.176495

2020

DOI: 10.1101/2020.06.28.176495

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Conserved Role of the Large Conductance Calcium-Activated Potassium Channel, K_Ca1.1, in Sinus Node Function and Arrhythmia Risk

Santiago Pined

Vesna Nikolova-Krstevski

Christiana Leimena

et al.

Abstract: Background -KCNMA1 encodes the α-subunit of the large-conductance Ca 2+ -activated K +

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Conserved Role of the Large Conductance Calcium-Activated Potassium Channel, K_Ca1.1, in Sinus Node Function and Arrhythmia Risk

Abstract: Background -KCNMA1 encodes the α-subunit of the large-conductance Ca 2+ -activated K +

Cited by 0 publications

References 47 publications

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Conserved Role of the Large Conductance Calcium-Activated Potassium Channel, KCa1.1, in Sinus Node Function and Arrhythmia Risk

Abstract: Background -KCNMA1 encodes the α-subunit of the large-conductance Ca 2+ -activated K +

Cited by 0 publications

References 47 publications

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Conserved Role of the Large Conductance Calcium-Activated Potassium Channel, K_Ca1.1, in Sinus Node Function and Arrhythmia Risk