Conners' Rating Scales - Revised

Conners, C. Keith

doi:10.1037/t04967-000

Cited by 723 publications

(1,053 citation statements)

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“…Other outcomes included caregiver and teacher ratings of behavior (Conners' Rating Scale Revised: Long Version Global Index, comprising Restless-Impulsive and Emotional Lability factor sets [caregiver-rated T scores range from 38 to 90, and teacher-rated T scores range from 40 to 90, with higher scores indicating worse functioning]), 12 and the Behavior Rating Inventory of Executive Function [BRIEF] Global Executive Composite T score, comprising summary measures of behavioral regulation and metacognition [caregiver-rated scores range from 28 to 101, and teacher-rated scores range from 37 to 131, with higher scores indicating worse functioning]) 13 ; symptoms of the obstructive sleep apnea syndrome, as assessed by means of the Pediatric Sleep Questionnaire sleep-related breathing disorder scale (PSQ-SRBD), in which scores range from 0 to 1, with higher scores indicating greater severity 14 ; sleepiness, as assessed with the use of the Epworth Sleepiness Scale modified for children, in which scores range from 0 to 24, with higher scores indicating greater daytime sleepiness 15 ; global quality of life (caregiver-rated total score from the Pediatric Quality of Life Inventory [PedsQL], in which scores range from 0 to 100, with higher scores indicating better quality of life) 16 ; disease-specific quality of life (total score on the 18-item Obstructive Sleep Apnea-18 assessment tool, in which scores range from 18 to 126, with higher scores indicating worse quality of life) 17 ; generalized intellectual functioning (General Conceptual Ability score from the Differential Ability Scales-II [DAS], in which scores range from 30 to 170, with higher scores indicating better functioning) 18 ; and polysomnographic indexes.…”

Section: Discussionmentioning

confidence: 99%

A Randomized Trial of Adenotonsillectomy for Childhood Sleep Apnea

Marcus

Moore

Rosen

et al. 2014

Survey of Anesthesiology

229

438

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BACKGROUND-Adenotonsillectomy is commonly performed in children with the obstructive sleep apnea syndrome, yet its usefulness in reducing symptoms and improving cognition, behavior, quality of life, and polysomnographic findings has not been rigorously evaluated. We hypothesized that, in children with the obstructive sleep apnea syndrome without prolonged oxyhemoglobin desaturation, early adenotonsillectomy, as compared with watchful waiting with supportive care, would result in improved outcomes.

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Section: Discussionmentioning

confidence: 99%

A Randomized Trial of Adenotonsillectomy for Childhood Sleep Apnea

Marcus

Moore

Rosen

et al. 2014

Survey of Anesthesiology

229

438

View full text Add to dashboard Cite

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“…This information was supplemented with two standardized questionnaires: the Conners Parent and Teacher Rating ScalesRevised [Conners, 1997] and Ontario Child Health Survey Scales-Revised [Boyle et al, 1993]. Subjects were excluded if there was evidence of neurological or chronic medical illness, bipolar affective disorder, psychotic symptoms, Tourette syndrome or chronic multiple tics.…”

Section: Family-based Association Analysismentioning

confidence: 99%

A family‐based association analysis and meta‐analysis of the reading disabilities candidate gene DYX1C1

Tran

Gagnon

Wigg

et al. 2013

American J of Med Genetics Pt B

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Reading disabilities (RD) have a significant genetic basis and have shown linkage to multiple regions including chromosome 15q. Dyslexia susceptibility 1 candidate gene 1 (DYX1C1) on chromosome 15q21 was originally proposed as a candidate gene with two potentially functional polymorphisms at the À3G/A and 1249G/T positions showing association with RD. However, subsequent studies have yielded mixed results. We performed a literature review and meta-analysis of the À3G/A and 1249G/T polymorphisms, including new unpublished data from two family-based samples. Ten markers in DYX1C1 were genotyped in the two independently ascertained samples. Single marker and À3G/A:1249G/T haplotype analyses were performed for RD in both samples, and quantitative trait analyses using standardized reading-related measures was performed in one of the samples. For the meta-analysis, we used a random-effects model to summarize studies that tested for association between À3G/A or 1249G/T and RD. No significant association was found between the DYX1C1 SNPs and RD or any of the reading-related measures tested after correction for the number of tests performed. The previously reported risk haplotype (À3A:1249T) was not biased in transmission. A total of 9 and 10 study samples were included in the meta-analysis of the À3G/A and 1249G/T polymorphisms, respectively. Neither polymorphism reached statistical significance, but the heterogeneity for the 1249G/T polymorphism was high. The results of this study do not provide evidence for association between the putatively functional SNPs À3G/A and 1249G/T and RD. Ó 2013 Wiley Periodicals, Inc.Key words: dyslexia; chromosome 15q; EKN1; genetics INTRODUCTIONSpecific reading disabilities (RD), or developmental dyslexia, refer to an unexpected and specific difficulty in learning to read despite normal or above average intelligence, education, and socioeconomic opportunity. RD is likely caused by a dysfunction of neural systems involved in cognitive skills required for reading [Habib, 2000]. A core deficit observed in dyslexics involves the processing of phonemes, the most basic unit of speech sounds, which persist into adulthood [Bruck, 1992;Shaywitz et al., 1999;Ramus et al., 2003]. Individuals with RD have difficulty segmenting words into their phonological elements, impairing their ability in learning to read. Several other reading components have been shown to be impaired in individuals with RD including orthographic coding, single-word reading, rapid automatic naming, and spelling [Denckla and Rudel, 1976;Paulesu et al., 2001;Francks et al., 2002]. With an estimated Additional supporting information may be found in the online version of this article. 146Neuropsychiatric Genetics prevalence ranging from 5% to 17.5% in school-aged children, RD is the most common learning disability and affects four out of five individuals with learning disabilities [Shaywitz et al., 1990;Shaywitz, 1998;Katusic et al., 2001]. There is substantial evidence to suggest that RD is both familial and heritable. RD has bee...

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“…ADHD-CT diagnosis was made using the Parental Account of Childhood Symptoms interview (PACS) 24 and the ADHD subscale of the Conners Teacher Rating Scale Revised Long Version (CTRS-R: L), 25 using a procedure which has been described in full previously 23 .…”

Section: Methodsmentioning

confidence: 99%

“…Conduct problems (CP) were measured using the relevant subscales of the parent form of the Conners Rating Scales-Revised Long Version (CRS-R: L) 25 including ODD and CDrelated symptoms each rated on a 4-point Likert scale. Emotional problems (EMO) were measured using the parent form of the Strengths and Difficulties Questionnaire (SDQ) 27 including 5 items scored on a 3-point Likert scale.…”

mentioning

confidence: 99%

Disentangling Child and Family Influences on Maternal Expressed Emotion Toward Children With Attention-Deficit/Hyperactivity Disorder

Cartwright¹,

Bitsakou²,

Daley³

et al. 2011

Journal of the American Academy of Child & Adolescent Psych

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Objective: We used multi-level modelling of sib-pair data to disentangle the influence of child-specific and shared family influences on maternal expressed emotion (MEE) towards children and adolescents with attentiondeficit/hyperactivity disorder (ADHD). Method: MEE was measured for 60 ADHD proband-unaffected sibling pairs aged 6-17 years using the Five Minute Speech Sample. Questionnaire measures of conduct and emotional problems were collected for children and measures of depression and ADHD were collected for mothers. Multilevel models partitioned the effects of 5 MEE components (Initial Statement -IS, Relationship -REL, Warmth -WAR, Critical Comments -CC and Positive Comments -PC) into (i) child-specific and (ii) shared family effects. Results: Significant child-specific influences were confirmed for all MEE components, with higher levels of MEE expressed toward probands than siblings. For REL, PC, and CC, this effect was explained by comorbid conduct problems rather than ADHD. Only low WAR was associated with ADHD itself. Furthermore, only low WAR showed shared familial influences which were in part accounted for by maternal depression.Conclusions: MEE towards ADHD children was influenced by child-specific factors. For most components these were driven by comorbid symptoms of conduct problems rather than ADHD itself. WAR was different -it was influenced by both child-specific and shared familial effects and the child effects showed no relation to conduct problems. Further studies utilising a longitudinal design are required to establish the direction of causation and extend our understanding of the relationship between EE components and ADHD.

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Conners' Rating Scales - Revised

Cited by 723 publications

References 0 publications

A Randomized Trial of Adenotonsillectomy for Childhood Sleep Apnea

A Randomized Trial of Adenotonsillectomy for Childhood Sleep Apnea

A family‐based association analysis and meta‐analysis of the reading disabilities candidate gene DYX1C1

Disentangling Child and Family Influences on Maternal Expressed Emotion Toward Children With Attention-Deficit/Hyperactivity Disorder

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