Compression of the сeliac trunk syndrome. History of the problem

Abstract

The article presents the main stages of the development of ideas about a fairly rare pathology in clinical practice the syndrome of compression of the ventral trunk. Until the mid-nineteenth century, reports of the clinical picture of chronic abdominal ischemia were more descriptive. Serial pathoanatomic studies, comparison of anatomical features revealed at autopsy with clinical manifestations accompanying these changes, contributed to the isolation of chronic ischemic disease of the digestive system in a separate nosological form. The discovery of angiography contributed to the significant development of vascular surgery and the isolation of the ventral trunk compression syndrome as an independent disease, since it allowed for a lifetime comparison of the detected changes in blood vessels with the clinical manifestations of the disease. The term ventral trunk compression syndrome was first introduced by P. Harjola when describing a case of abdominal pain in a patient with compression of the ventral trunk by the neuroganglionic tissue of the ventral plexus. Later, J. Dunbar confirmed the Association of clinical manifestations of chronic abdominal pain with compression of the ventral trunk by the median arched ligament of the diaphragm. In English-language literature, the disease was named Dunbar syndrome. The effectiveness of eliminating the symptoms of this disease by surgical dissection of compression structures has been proven. Further study of this problem developed in parallel with the development of surgery in General in accordance with the emergence and introduction of new surgical technologies. The purpose of numerous studies was to study the etiopathogenetic mechanisms of the occurrence of a variety of clinical manifestations of this syndrome and improve the results of its treatment by improving known surgical techniques and introducing new ones.

Celiac Artery Compression Syndrome: A Retrospective Single-Center Analysis of Diagnosis and Surgical Treatment

“…В настоящее время ХМИ определяется как ишемические симптомы (абдоминальная боль после приема пищи, снижение массы тела в результате отказа от еды и, собственно, развитие страха боли от приема пищи), вызванные недостаточностью мезентериального артериального кровоснабжения внутрисосудистой или внесосудистой этиологии. Термин «синдром компрессии чревного ствола» (СКЧС) как описание изолированного окклюзионно-стенотического поражения ЧС, возникающего вследствие экстравазального сдавления артерии окружающими структурами, был впервые предложен в середине XX столетия [1][2][3][4][5].…”