Complete testicular infarction secondary to epididymoorchitis and pyocele

Epididymo-Orchitis Leading to Global Testicular Infarction in a Pediatric Patient – A Case Report

“…4 This association with urinary/coliform pathogens appears to be consistent for cases of testicular infarction in EO as well, and many cases reported in the literature had urine cultures positive for E. coli. 2,3,[7][8][9][10] History of trauma has not been previously reported to be associated with this complication, and it is unclear whether the scrotal trauma that occurred in our patient's case expedited the infectious or ischemic process, or was simply coincidental.…”

“…In contrast, absent Doppler flow combined with areas of hypoechoic tissue within the testicle is highly suggestive of infarction in the setting of EO. 9 If standard ultrasound is inconclusive, microbubble contrast-enhanced ultrasonography can more definitively determine the presence of testicular hypoperfusion. 10 If able to be performed rapidly, scrotal MRI is another option that can more obviously demonstrate lack of perfusion with a specificity approaching 100%.…”

Neonatal pyocele originating from a urinary tract infection: a case report

“…Even though most cases of pyocele are idiopathic in etiology and the source of infection remains unrecognized, there have been descriptions of various possible mechanisms of pathogenesis in the literature [ 1 , 6 – 8 , 10 ]. These mechanisms include hematogenous seeding of the serosa lining, secondary infection of a hydrocele following orchitis or epididymitis, seeding from an intraperitoneal source through a patent tunica vaginalis, or reflux of urine into the vas deferens, which are the most common pathways at fault [ 3 , 12 , 13 ].…”

“…Moreover, the pathogens in most reported cases were found to be bacterial, primarily Escherichia coli ( E. coli ) and, in some instances, Staphylococcus lugdunensis , Klebsiella pneumonia , β-hemolytic streptococci, Salmonella spp., Bacteroides fragilis , and Proteus mirabilis [ 2 – 6 , 9 ]. However, we found no reports in the literature of the spread of the infection from the urinary tract [ 3 , 9 , 10 ]. Therefore, we report a case of a 23-day-old boy with pyocele and a confirmed urinary tract infection.…”

Ultrasound Diagnostic of Testicular ischemia without torsion in children. Rare clinical observations

“…описывают повреждение тестикулярной ткани в виде гипоэхогенных, реже гиперэхогенных, очагов клиновидной или округлой формы в одном из отделов яичка (чаще в верхнем полюсе яичка) [8,10,14]. В ранние сроки (первые 6 ч) выраженных изменений в В-режиме может и не быть, но в режиме ЦДК определяется резкое ослабление/отсутствие интратестикулярного сосудистого рисунка (Se = 85-90 %, Sp = 100 %) [1,12]. В собственном клиническом наблюдении № 1 сосудистый рисунок в яичке регистрировался в виде единичных цветовых пикселей, и было зафиксировано резкое повышение показателей периферического сопротивления в интратестикулярных артериях (RI = 1,05), что соответствует данным литературы о подобных наблюдениях [1,12].…”