2014
DOI: 10.4103/0973-1482.131375
|View full text |Cite
|
Sign up to set email alerts
|

Comparison of nucleostemin gene expression in CD133+ and CD133− cell population in colon cancer cell line HT29

Abstract: It is concluded that nucleostemin expression could not be specific in a certain type of cells in colon cancer cell line HT29 and controlling strategies in colon cancer must not be focused on one certain type of colon cancer cells as main expressing nucleostemin gene.

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
1

Citation Types

0
1
0

Year Published

2017
2017
2022
2022

Publication Types

Select...
2

Relationship

0
2

Authors

Journals

citations
Cited by 2 publications
(1 citation statement)
references
References 28 publications
0
1
0
Order By: Relevance
“…GNL3 was initially named nucleostemin (NS) ( 17 ). GNL3 was subsequently shown to play significant roles in regulating cell proliferation and the cell cycle in various human cancers, such as gastric cancer, colorectal cancer, liver cancer and other malignancies ( 19 , 20 ). Furthermore, GNL3 also plays a crucial role in many other physiological and pathological processes, such as the resistance to apoptosis and cellular aging, cellular self-renewal, the maintenance of stemness, the inhibition of differentiation, ribosome biogenesis, the maintenance of genome stability and telomere integrity, radioresistance, the maintenance of tumor-initiating cells, and the induction of pluripotent stem cells ( 21 27 ).…”
Section: Introductionmentioning
confidence: 99%
“…GNL3 was initially named nucleostemin (NS) ( 17 ). GNL3 was subsequently shown to play significant roles in regulating cell proliferation and the cell cycle in various human cancers, such as gastric cancer, colorectal cancer, liver cancer and other malignancies ( 19 , 20 ). Furthermore, GNL3 also plays a crucial role in many other physiological and pathological processes, such as the resistance to apoptosis and cellular aging, cellular self-renewal, the maintenance of stemness, the inhibition of differentiation, ribosome biogenesis, the maintenance of genome stability and telomere integrity, radioresistance, the maintenance of tumor-initiating cells, and the induction of pluripotent stem cells ( 21 27 ).…”
Section: Introductionmentioning
confidence: 99%