Comparison of Doppler echocardiography, color M-mode Doppler, and Doppler tissue imaging for the estimation of pulmonary capillary wedge pressure

González‐Vílchez, Francisco; Ayuela, José; Ares, Miguel; Mata, Nuria Sánchez; González, Amparo García; Durán, Rafael

doi:10.1067/mje.2002.125752

Cited by 68 publications

(63 citation statements)

References 34 publications

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“…Other methods have been developed to improve the estimates of diastolic function, including pulse-wave Doppler at the PV ostia (PV velocity measurements), 32,33 tissue Doppler measurement of ventricular relaxation, 34 -36 color Doppler M-mode across the mitral valve (propagation velocity), [37][38][39] and speckled tracking analysis (strain rates). 40 -42 In addition, other groups have used more indirect measures, such as plasma brain natriuretic peptide levels [43][44][45] or LA size, 46 to enhance the picture of diastolic function.…”

Section: Measurement Of Ventricular Diastolic Functionmentioning

confidence: 99%

Diastolic Dysfunction and Risk of Atrial Fibrillation

2012

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A trial fibrillation (AF) is the most common sustained arrhythmia, and its prevalence in the population is increasing. 1 Diastolic dysfunction shares many common risk factors with AF, including age, hypertension, 2-5 obesity, 6,7 and diabetes. 8,9 Like AF, diastolic dysfunction increases with age, 10 and patients given the diagnosis of diastolic dysfunction are more likely to have AF at the time. 11 Diastolic dysfunction has significant pathological effects on atrial structure and function, many of which are proarrhythmic. However, much remains to be learned about the specific mechanisms through which diastolic dysfunction ultimately promotes AF.Previous reviews have examined the broad association of diastolic dysfunction and AF. 12 In this review, we attempt to examine this association on a mechanistic level. We begin with a basic review of the physiology of diastolic function, with particular attention to the complex interaction between the atrium and the ventricle during diastole. We then provide an overview of some of the most common clinical methods to quantify diastolic function and highlight the strengths and weaknesses of these methods with regard to providing an accurate picture of this physiology. We describe how these methods are applied to diagnose diastolic dysfunction, including the development of a widely adopted classification scheme of diastolic dysfunction. We then review the limited clinical data available connecting diastolic dysfunction with the risk of incident, nonvalvular AF, with a focus on studies examining diastolic dysfunction in populations without structural heart disease-ie, with preserved systolic function in the absence of hypertrophic cardiomyopathic disease or congenital heart disease. Finally, we attempt to reconcile the results of these clinical studies with experimental data in both human, animal, and cellular models, to create a mechanistic link between diastolic dysfunction and pathological changes that increase the likelihood of AF. Atrial and Ventricular Diastolic DynamicsA number of excellent resources are available for more detailed review of the dynamics of ventricular diastole, including invasive 13 and noninvasive 14 -17 descriptions. Ventricular diastole is the period of the cardiac cycle that begins with the closure of the aortic valve and ends with mitral valve closure, during which the ventricle is filling with blood. It can be further divided into 4 sequential components: (1) isovolumic relaxation, (2) early rapid filling, (3) diastasis, and (4) atrial systole (late filling). Dynamically, none of the components is completely independent of each other, or the systolic function of the heart, and changes in one can influence the others in predictable and unpredictable ways.Essentially, there are 2 characteristics of ventricular function that contribute to diastolic function: relaxation (lusitropy) and compliance. Relaxation refers to the process by which the ventricular pressure drops after contraction to a level below that of the atrium, causing blood to flow dow...

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Section: Measurement Of Ventricular Diastolic Functionmentioning

confidence: 99%

Diastolic Dysfunction and Risk of Atrial Fibrillation

2012

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“…However, ambiguous outcomes can be attained due to preload dependency of some of these measurements [20,31]. This has subsequently led to the introduction of the flow propagation velocity measure, V p , as the diastolic relaxation index due to its preload-independent characteristics [11,14,23].…”

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confidence: 99%

Left ventricular flow propagation velocity measurement: Is it cast in stone?

Chan

Yeoh

Liew

et al. 2017

Med Biol Eng Comput

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“…In our study, we also appraised a newer parameter, E/Vp, indicating LV diastolic functions and filling pressures. The ratio of E to Vp was shown to be an informative parameter in patients especially with low ejection fraction and comparable with E/E' ratio and Vp alone (25)(26)(27)(28)(29). Before and after infusion E to Vp ratio was not significantly different between the two groups, levosimendan improved E/Vp almost similarly in both groups (Table 2).…”

Section: Discussionmentioning

confidence: 70%

Efficacy of levosimendan in patients with chronic heart failure: Does rhythm matter?

Yontar¹,

Yılmaz²,

Yalta³

et al. 2010

Anadolu Kardiyol Derg

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Amaç: Levosimendan, kardiyak kontraktiliteyi arttıran yeni jenerasyon inotropik bir ajandır. Diğer inotropiklerin aksine, levosimendan miyokart hücresinin kalsiyum alımını arttırmadığı için, kalsiyum yüklenmesine ve ilişkili aritmilere sebep olmaz. Atrial fibrilasyonun (AF) kalp yetersizliğin-de (KY) mortalite ve morbidite için bağımsız risk faktörü olduğu geniş popülasyonlu çalışmalarla gösterilmiştir. Kalp yetersizliği, AF sıklığını arttırır, AF ise KY'yi kötüleştirir, dolayısı ile genellikle birliktelik gösterirler. Çalışmamızda levosimendan uygulamasının AF ve sinüs ritmindeki hastalarda sistolik-diyastolik fonksiyonlara etkisini karşılaştırdık. Yöntemler: Çalışma prospektif tipinde dizayn edildi. Akut dekompanse, sistolik disfonksiyona bağlı KY nedeniyle yatırılan ve levosimendan verilmesi planlanan hastalar çalışmaya alındı. Hastalarda AF olup olmamasına göre iki gruba ayrıldılar (grup A ve S). Uygulama öncesi ve sonrası hastaların ekokardiyografik incelemeleri yapıldı. Ekokardiyografik veriler ANOVA tekrarlanan ölçümler testiyle değerlendirildi. Bulgular: Bazal sol ventrikül ejeksiyon fraksiyonu (SVEF) AF grubunda daha kötüydü (ortalama SVEF, grup A:%20.9, grup S:%26.4, p=0.04). Bazal diyastolik parametreler eşit derecede bozulmuştu, uygulama sonrasında deselerasyon süresi (DT) haricinde, izovolumik kontraksiyon zamanı (IVRT) ve yayılım hızı (Vp) benzer ölçülerde düzelme gösterdi. İnfüzyon sonrası, IVRT değerleri hem grup S'de (108.6±23.2 msn'den 100.4±28.4 msn'ye) hem de grup A'da (117.3±25.1msn'den 92.0±20.9 msn'ye) gruplar arası anlamlı fark olmadan düzelme gösterdi (p=0.012 ve etkileşim için p=0.180). Bir diğer önemli diyastolik parametre olan Vp de iki grupta da benzer (etkileşim için p=0.498) ölçüde düzeldi (grup A için, 35.4±8.8 cm/ sn'den 41.1±7.7 grup S için, 33.7±7.5 cm/sn'den 37.8±7.6 cm/sn'ye, p<0.01). ÖZETOb jec ti ve: Levosimendan is a relatively new inotropic agent. Unlike other inotropic agents, Levosimendan does not increase cellular calcium intake, so that, does not cause intracellular calcium overload and related arrhythmias. Atrial fibrillation (AF) was shown to be an independent risk factor for mortality and morbidity in large heart failure (HF) trials. Heart failure induces AF, AF aggravates HF and therefore they generally coexist. We conducted a study to investigate if there is any differential effect of Levosimendan in HF patients with chronic AF and without AF. Methods: This is a prospective study. Consecutive patients, who were hospitalized because of acutely decompensated HF due to systolic dysfunction and decided Levosimendan administration, were enrolled. Patients were classified into two as those with AF (group A) and those with sinus rhythm (control group, group S). All patients had echocardiography before and after administration. Echocardiographic data were evaluated by ANOVA repeated measurements test. Results: Baseline left ventricle ejection fraction (LVEF) was poorer in group with AF (mean LVEF for group A: 20.9%, for group S: 26.4%, p=0.04). Baseline diastolic paramet...

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Comparison of Doppler echocardiography, color M-mode Doppler, and Doppler tissue imaging for the estimation of pulmonary capillary wedge pressure

Cited by 68 publications

References 34 publications

Diastolic Dysfunction and Risk of Atrial Fibrillation

Diastolic Dysfunction and Risk of Atrial Fibrillation

Left ventricular flow propagation velocity measurement: Is it cast in stone?

Efficacy of levosimendan in patients with chronic heart failure: Does rhythm matter?

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