1995
DOI: 10.1016/0016-5085(95)24326-7
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Comparative studies of epidermal growth factor, epidermal growth factor receptor, K-ras, and P53 in gallbladder carcinomas with and without an anomalous junction of pancreticobiliary duct

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“…Activation of KRAS can induce the constitutive activation of the MAPK pathway and PI3K-AKT pathway, which rapidly develops the growth of the gallbladder epithelium (Leevers and Marshall, 1992;Thomas et al, 1992;Rubio et al, 1997). Furthermore, point mutation of P53 is also related to gallbladder cancer carcinogenesis (Hanada et al, 1997;Watanabe et al, 1999). Therefore, U0126 as a MAPK inhibitor has the potential to inhibit gallbladder cancer proliferation.…”
Section: U0126 and Gallbladder Cancermentioning
confidence: 99%
“…Activation of KRAS can induce the constitutive activation of the MAPK pathway and PI3K-AKT pathway, which rapidly develops the growth of the gallbladder epithelium (Leevers and Marshall, 1992;Thomas et al, 1992;Rubio et al, 1997). Furthermore, point mutation of P53 is also related to gallbladder cancer carcinogenesis (Hanada et al, 1997;Watanabe et al, 1999). Therefore, U0126 as a MAPK inhibitor has the potential to inhibit gallbladder cancer proliferation.…”
Section: U0126 and Gallbladder Cancermentioning
confidence: 99%