Abstract:See original paper on page 893 F amilial renal glucosuria resulting from a defect in the proximal tubular sodium-dependent glucose cotransporter (SGLT)2 is regarded as an innocent condition without associated morbidity [1,2]. To alleviate hyperglycemia in type 2 diabetes, for instance in the setting of obesity and hypertension, the induction of renal glucosuria by SGLT(2) inhibition therefore appears an interesting therapeutic option [3], which, importantly, does not require insulin. SGLT2 mediates the co-tran… Show more
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