Combined Apical Hypertrophic Cardiomyopathy and Coronary Artery-Left Ventricular Fistula

Chia, B.L.; Yan, Peter; Choo, Maurice H.

doi:10.1378/chest.94.1.219b

Cited by 8 publications

(3 citation statements)

References 5 publications

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“…The coronary anomalies are thought to be due to sheer stress-induced alteration of fetal vascular gene expression, disturbing the development of proximal coronaries and their connection with the coronary orifices within the aortic sinuses 5. Microfistulae in keeping with vessels of Wearn have also been described in association with hypertrophic cardiomyopathy,6 although the causative mechanism is unknown.…”

Section: Discussionmentioning

confidence: 99%

Association of coronary to left ventricular microfistulae (vessels of Wearn) with atrial septal defect in an adult without cyanotic heart disease

Hussain¹,

Roberts

2015

BMJ Case Reports

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Vessels of Wearn are rare findings during coronary angiography in adults. They are known to be associated with forms of cyanotic congenital heart disease in infants but we are not aware of any published cases of association with non-cyanotic left to right shunts in adults. We present the case of a 69-year-old man with angiographically evident vessels of Wearn draining from the left and right coronary arteries into the left ventricle associated with an asymptomatic atrial septal defect. We postulate a developmental phase association between atrial septal maturation and closure of perfusing microchannels from the ventricular cavities to the epicardial coronary arteries on the same spectrum as that which leads to more widespread defects in infants. We also highlight a common medication side effect that might have been mistaken as a manifestation of the congenital anomalies.

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Section: Discussionmentioning

confidence: 99%

Association of coronary to left ventricular microfistulae (vessels of Wearn) with atrial septal defect in an adult without cyanotic heart disease

Hussain¹,

Roberts

2015

BMJ Case Reports

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“…Esta formação aneurismática geralmente surge após o infarto do miocárdio 8 , pode ser devida a obstrução intracavitária médio-ventricular com gradiente de pressão intraventricular que facilita a formação do aneurisma, pelo aumento da pressão apical e/ ou com a redução da pressão de perfusão coronária desta região 21,22 , porém pode não estar relacionada a áreas de infarto 23 ou não estar relacionada a lesão obstrutiva coronária 8 . Foi observado também a associação entre a angina do peito e a presença de fístulas arteriovenosas coronárias em pacientes com CMHA [24][25][26] , ambas as condições poderão ser responsáveis pelos sintomas anginosos, podendo também as fístulas serem causadoras de roubo coronário. Nos casos com grande defeito na perfusão e resposta positiva ao esforço com tálio, estará indicada a correção cirúrgica das fístulas [24][25][26] .…”

Section: Discussionunclassified

“…Foi observado também a associação entre a angina do peito e a presença de fístulas arteriovenosas coronárias em pacientes com CMHA [24][25][26] , ambas as condições poderão ser responsáveis pelos sintomas anginosos, podendo também as fístulas serem causadoras de roubo coronário. Nos casos com grande defeito na perfusão e resposta positiva ao esforço com tálio, estará indicada a correção cirúrgica das fístulas [24][25][26] . Nosso caso é de paciente que sabidamente era portador da forma apical há 12 anos, tendo sido inicialmente diagnosticado como portador de DAC, porém, como não respondeu à terapia com nitrato e propranolol, foi submetido a Referências estudo invasivo que mostrou coronárias normais e a hipertrofia da ponta do VE.…”

Section: Discussionunclassified

Cardiomiopatia hipertrófica apical associada a doença arterial coronária

Lopes

Diamant

et al. 1998

Arq. Bras. Cardiol.

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Atypical Roentgenographic Manifestations of Pneumocystis carinii Pneumonia

Kennedy¹,

Goetz²

1992

Arch Intern Med

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Although Pneumocystis carinii pneumonia (PCP) usually presents with bilateral interstitial pulmonary infiltrates, many other roentgenographic presentations occur in human immunodeficiency virus-infected patients. To clarify the determinants of atypical presentations of PCP, we evaluated 65 English-language reports that related the roentgenographic manifestations of consecutive cases of PCP. The incidence of PCP-associated upper lobe disease, cysts, and spontaneous pneumothoraxes was increased in human immunodeficiency virus-infected patients receiving aerosolized pentamidine prophylaxis. Normal chest roentgenograms were more common and nodular lesions were less common in human immunodeficiency virus-infected patients than in uninfected patients. However, the roentgenographic manifestations of PCP could not be specifically predicted by a patient's underlying disease. Neither zidovudine therapy nor intravenous drug use apparently affected the roentgenographic presentation of PCP. Unusual pathologic responses to PCP, including granuloma formation, vascular invasion, and microscopic foci of calcification, were present in all patient groups.

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Combined Apical Hypertrophic Cardiomyopathy and Coronary Artery-Left Ventricular Fistula

Cited by 8 publications

References 5 publications

Association of coronary to left ventricular microfistulae (vessels of Wearn) with atrial septal defect in an adult without cyanotic heart disease

Association of coronary to left ventricular microfistulae (vessels of Wearn) with atrial septal defect in an adult without cyanotic heart disease

Cardiomiopatia hipertrófica apical associada a doença arterial coronária

Atypical Roentgenographic Manifestations of Pneumocystis carinii Pneumonia

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