Ulvund, M. J.: Ovlne wbite-Iiver disease (OWLD). Trace elements in liver. Acta vet. scand. 1990,31,297-307. -Trace elements in liver were examined in vitamin BI2 deficient lambs which developed ovine white-liver disease (OWLD), in cobalt! vitamin BI2 supplemented lambs on the same pastures as well as clinically healthy, but sometimes subclinical BI2 deficient lambs on other pastures (H). Liver Co was marginal to deficient in both OWLD lambs (S lambs) and H lambs. Supplementation with BI2 or Co elevated liver Co. Liver copper was significantly lower in OWLD lambs than in the H lambs, and CO/BI2 supplementation on pasture generally had no significant effecton the contents. Dosing Iambs on OWLD pastures with copper oxide needles (SCuO), however, resulted in high/toxic liver Cu. Dosing with Co, Se and Cu glass boluses resulted in adequate liver Cu, except for I lamb with toxic amounts indicating dissolution and absorption of the bolus. OWLD lambs had significantly lower liver molybdenum than H lambs, and CO/BI2 supplementation elevated values, while CuO treatment depressed them. Liver zinc, manganese and selenium are also reported.sheep; cobalt/vitamin BI2 deficiency ; trace element interferences; copper.