Handbook of Interventions for Structural Heart and Peripheral Vascular Disease 2017
DOI: 10.5005/jp/books/12983_23
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Coarctation of Aorta

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Cited by 2 publications
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“…A frequent surgical treatment for conditions such as aortic constriction and aneurysms is end-to-end anastomosis [5,23]. Through this treatment, the aorta's blood flow channel is reshaped to enhance heart function and reduce pain.End-to-end anastomosis is one of the recommended treatments for aortic constriction in pediatric patients because of the high degree of flexibility of the aorta.…”
Section: Discussionmentioning
confidence: 99%
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“…A frequent surgical treatment for conditions such as aortic constriction and aneurysms is end-to-end anastomosis [5,23]. Through this treatment, the aorta's blood flow channel is reshaped to enhance heart function and reduce pain.End-to-end anastomosis is one of the recommended treatments for aortic constriction in pediatric patients because of the high degree of flexibility of the aorta.…”
Section: Discussionmentioning
confidence: 99%
“…In the 1970s, Dr. Steidel and his colleagues first mentioned this disease, which can lead to structural abnormalities of the heart and blood vessels, thereby affecting the normal fun ction of the body [4]. The term "CoA" typically describes a restrict ed constriction of the thoracic descending aorta, which is situated next to the patent ductus arteriosus connection and distal to th e left subclavian artery [5]. This disorder is more common and ca uses 5%-8% of congenital cardiac abnormalities [5].Conversely, IAA denotes a congenital aortic arch deformity wherein the ascendin g and descending aortas do not have a direct physical relationshi p. Unlike CoA, IAA is a real discontinuity phenomeon [6][7][8].…”
mentioning
confidence: 99%
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“…Coarctation of the aorta (CoA) occurs in 0.3 of every 1,000 live births (1) and makes up 5%-8% of all congenital heart diseases (2). It is likely to represent a spectrum of aortic narrowing from a discrete entity to tubular hypoplasia, with many variations seen between the two extremes (3).…”
Section: Introductionmentioning
confidence: 99%
“…The main pathophysiological change of CoA is hypertension proximal to coarctation, and the postulated mechanisms of hypertension are mechanical obstruction and renin-angiotensinmediated humoral mechanisms (2). The increased afterload of the left ventricle will cause left ventricular hypertrophy.…”
Section: Introductionmentioning
confidence: 99%