Co‐worker fatalities from hydrogen sulfide

Hendrickson, Robert G.; Chang, Arthur; Hamilton, Richard

doi:10.1002/ajim.10355

Cited by 119 publications

(76 citation statements)

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“…Job duty analysis showed that 25% of victims were doing repair or maintenance at the time of death [6]. In an additional study of occupational hydrogen sulfide deaths, Hendrickson et al retrospectively reviewed the United States Bureau of Labor Statistics (USBLS) database: the Census of Fatal Occupational Injuries (CFOI) [7]. In this 7 year study from 1993 to 1999, 42 hydrogen sulfide events were recorded, and they resulted in 52 deaths.…”

Section: What Is the Historical And Current Epidemiology Of H 2 S Poimentioning

confidence: 99%

“…In this 7 year study from 1993 to 1999, 42 hydrogen sulfide events were recorded, and they resulted in 52 deaths. There were 9 multiple victim events and 21% of all fatalities involved co-workers of an index case [7]. Forty-five of these deaths (87%) occurred at the scene [7].…”

Section: What Is the Historical And Current Epidemiology Of H 2 S Poimentioning

confidence: 99%

“…There were 9 multiple victim events and 21% of all fatalities involved co-workers of an index case [7]. Forty-five of these deaths (87%) occurred at the scene [7]. Most workers killed were white (85%), male (98%), and middle-aged (between 31-50 years).…”

Section: What Is the Historical And Current Epidemiology Of H 2 S Poimentioning

confidence: 99%

“…Most workers killed were white (85%), male (98%), and middle-aged (between 31-50 years). Additionally, duration of occupational experience appears to be a factor in mortality; death is more common in workers with less than 1 year of employment in their current duties [7]. In 2005, according to the American Association of Poison Control Centers, 1396 cases of H 2 S exposure were reported.…”

Section: What Is the Historical And Current Epidemiology Of H 2 S Poimentioning

confidence: 99%

“…The primary route of toxicity is through inhalation [1,[3][4][5][6][7][8][9][10][11][12][13][14][15][16]. Gastrointestinal and dermal absorption is negligible [14,16,17].…”

Section: What Are the Clinical Signs And Symptoms Of H 2 S Poisoning?mentioning

confidence: 99%

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Case files of the university of cincinnati fellowship in medical toxicology: Two patients with acute lethal occupational exposure to hydrogen sulfide

Policastro

Otten

2007

J. Med. Toxicol.

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We are presenting cases of 2 employees of a chemical plastics manufacturing plant who experienced acute, brief, inhalational exposure to lethal concentrations of hydrogen sulfide in a confined space. Eight other coworkers not present in the immediate exposure area also presented to local health care facilities with mild complaints of headache, nausea, and dizziness, but they suffered no significant sequelae.Patient #1 was a 55-year-old man who, while working in a confined space, uncoupled a nozzle from a steam pipe, collapsed within one minute, and suffered cardiopulmonary arrest. He had a prior history of asthma, hyperlipidemia, coronary artery disease, and was taking lisinopril, aspirin, and lipitor. Following chest compressions and rescue breathing, both by on-scene responders and EMS, the patient's blood pressure was 210/110 mmHg, the pulse was 100 bpm, and ventilation was assisted at 14/minute. On examination, the patient was minimally combative, with a Glasgow coma score of 6 (E1, V1, M4), with pupils 3 mm bilaterally and reactive to light. The patient had a small partial-thickness burn to the right chest, presumed to be a thermal burn from steam or the hot pipe. No cyanosis or pulmonary crackles were noted. Because of respiratory distress, the patient was intubated on-scene using fentanyl, diazepam, and vecuronium by the combined nurse-physician Air EMS flight team. Post-intubation vital signs were as follows: blood pressure 130/69 mmHg, pulse 95 bpm, respirations 14/min (assisted), and oxygen saturation 98% on 100% FiO 2 . The patient's temperature on arrival to the Emergency Department (ED) was 98.2°F (36.8°C), where his examination remained unchanged.Diagnostic studies were obtained in the ED. Arterial blood gas analysis on 100% O 2 showed: pH 7.24, pCO 2 58 mmHg, pO 2 284 mmHg, oxyhemoglobin 97.2%, deoxyhemoglobin 2.0%, methemoglobin 0.3%, and carboxyhemoglobin 0.0%. The blood appeared red. An electrocardiogram revealed a narrow complex tachycardia, without Q waves, ST-segment abnormalities, or interval changes. Serum chemistries were as follows: a sodium of 139 mEq/L, potassium of 4.2 mEq/L, chloride of 105 mEq/L, bicarbonate of 25 mEq/L, BUN of 15 mg/dL, creatinine of 0.9 mg/dL, and glucose of 156 mg/dL. Creatinine kinase (CK) was 299 IU/L, CK-MB 2.2 IU/L, and troponin-I was 0.05 IU/L. A complete blood count showed: white blood cells at 9.0 k/mm 3 , hemoglobin at 13.0 g/dL, hematocrit at 38.2%, and platelets at 130/mm 3 . Coagulation studies were normal, and a chest radiograph showed adequate endotracheal tube placement, absence of pulmonary edema, and a widened mediastinal silhouette, likely due to projection artifact on an antero-posterior film.Patient #2 was a 53-year-old male co-worker, with a history of hypertension, but he was not receiving medical therapy. He was

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Section: What Is the Historical And Current Epidemiology Of H 2 S Poimentioning

confidence: 99%

Section: What Is the Historical And Current Epidemiology Of H 2 S Poimentioning

confidence: 99%

Section: What Is the Historical And Current Epidemiology Of H 2 S Poimentioning

confidence: 99%

Section: What Is the Historical And Current Epidemiology Of H 2 S Poimentioning

confidence: 99%

“…The primary route of toxicity is through inhalation [1,[3][4][5][6][7][8][9][10][11][12][13][14][15][16]. Gastrointestinal and dermal absorption is negligible [14,16,17].…”

Section: What Are the Clinical Signs And Symptoms Of H 2 S Poisoning?mentioning

confidence: 99%

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