2001
DOI: 10.1046/j.1468-2982.2001.00263.x
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Cluster-like headache. A case secondary to the subclavian steal phenomenon

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Cited by 7 publications
(14 citation statements)
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“…Our hypothesis is based on a contralateral mass effect over left cranial nerves, mainly through traction of meningeal structures which might produce left cranial nerve enlargement, thus leading to: (1) An activation of the parasympathetic system through pressure over the VIIth cranial nerve: the parasympathetic outflow runs from the superior salivary nucleus via the facialnerve, through the sphenopalatine ganglion; 12 lacrimal and nasal glands are innervated by parasympathetic referents, which lead to the classical autonomic signs typical of trigeminal autonomic cephalalgias -lacrimation, reddening of the eye and nasal congestion, as well as a local thirdorder sympathetic nerve lesion secondary to carotid swelling. 12 (2) The trigeminal nerve itself is irritated by contralateral meningeal traction. The ophthalmic division is implicated in the reflex activation of the cranial parasympathetic outflow, via a functional reflex between caudal nucleus of the trigeminal vascular system and the superior salivary nucleus.…”
Section: Discussionmentioning
confidence: 99%
See 1 more Smart Citation
“…Our hypothesis is based on a contralateral mass effect over left cranial nerves, mainly through traction of meningeal structures which might produce left cranial nerve enlargement, thus leading to: (1) An activation of the parasympathetic system through pressure over the VIIth cranial nerve: the parasympathetic outflow runs from the superior salivary nucleus via the facialnerve, through the sphenopalatine ganglion; 12 lacrimal and nasal glands are innervated by parasympathetic referents, which lead to the classical autonomic signs typical of trigeminal autonomic cephalalgias -lacrimation, reddening of the eye and nasal congestion, as well as a local thirdorder sympathetic nerve lesion secondary to carotid swelling. 12 (2) The trigeminal nerve itself is irritated by contralateral meningeal traction. The ophthalmic division is implicated in the reflex activation of the cranial parasympathetic outflow, via a functional reflex between caudal nucleus of the trigeminal vascular system and the superior salivary nucleus.…”
Section: Discussionmentioning
confidence: 99%
“…Primary cluster headache is a well-defined entity, with no intracranial lesion associated. Nevertheless, cluster-like headache has been reported in posterior circulation disfunction [1][2][3] and other cerebrovascular pathologies, 4,5 cervical lesions 6 and other malignancies, 7,8 malformations, 9 or head trauma. 10 It has been established that in around 3-5% of patients, this syndrome is secondary to diverse cranial structural abnormalities, 11 mainly if atypical features, like absence of periodicity and regularly hourly recurrence, or neurological signs other than ptosis or miosis, are present.…”
Section: Introductionmentioning
confidence: 99%
“…Universidade Federal do Rio de Janeiro , for its part, researches spreading depression in chicken retina and neuroimaging. University of Parana carries out ongoing research into the mechanisms underlying migraine aura and neuropharmacology . The Federal University of Pernambuco has contributed to the study of headache for almost half a century since the first publication in 1968 .…”
Section: Publications In Headache Medicinementioning
confidence: 99%
“…University of Parana carries out ongoing research into the mechanisms underlying migraine aura and neuropharmacology. [244][245][246][248][249][250]255,256,[318][319][320][321][322][323][324][325][326][327][328] The Federal University of Pernambuco has contributed to the study of headache for almost half a century since the first publication in 1968. 40 Wilson Farias da Silva, 44 80 are a few of the researchers that have been involved in the study of different aspects of the diagnosis, physiopathology, and treatment of headache disorders in the Federal University of Pernambuco.…”
Section: Publications In Headache Medicinementioning
confidence: 99%
“…An extended review of 156 symptomatic, cluster-like headache cases showed that the more frequent pathologies mimicking primary cluster headache were vascular disorders, followed by tumors and inflammatory infectious diseases [2]. Various vascular pathology involving trigeminovascular system can trigger CLH: pseudoaneurysm within cavernous sinus [3], dural fistula [4], carotid artery dissection [5][6][7][8], arteriovenous malformation [9] and subclavian steal syndrome [10].…”
Section: Introductionmentioning
confidence: 99%