“…There was no mutation in any common AML prognostic gene, including FLT3, dupMLL, IDH1, IDH2, NPM1, KIT, NRAS, CEBPA, DNMT3A, PHF6, TET2, ASXL1, RUNX1, TP53, and WT1. Chromosome karyotypic analysis showed 46, XX, add (7, 11), t (15;17) (q24; q21) [6]/46, XX [14] (Figure 2). Subsequently, the patient underwent all-trans retinoic acid and arsenic acid induction therapy.…”