Clinical evaluation of Dinamap 845 automated blood pressure recorder.

Silas, J H; Barker, Anthony T.; Ramsay, LE

doi:10.1136/hrt.43.2.202

Cited by 128 publications

(39 citation statements)

References 5 publications

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“…9 *" Arterial blood pressure was measured automatically every 2 minutes in the left arm with an oscillometric blood pressure recorder (Dinamap, Model 845, Critikon, Tampa, FL, USA). 12 The same values were found for blood pressure in the left arm and in the right arm. Heart rate (HR) was determined using a continuous electrocardiographic monitor (Electronics for Medicine, Honeywell, Pleasantville, NY, USA).…”

Section: Methodssupporting

confidence: 69%

“…12 Interestingly, in previous studies 3 we observed that BA diameter was reduced after dihydralazine or cadralazine treatment, in contrast to the changes observed in response to other vasodilators, such as nitroglycerin or calcium inhibitors, which increased BA diameter. 4 Pharmacological research 1 ' 2 and previous studies from our laboratory 3 ' i have suggested that activation of the sympathetic nervous system leads to active vasoconstriction of the BA, in addition to inducing passive vasoconstriction following reduction in blood pressure.…”

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confidence: 80%

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Hemodynamics of the carotid artery after vasodilation in essential hypertension.

et al. 1988

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SUMMARYWe performed simultaneous noninvasive measurements of common carotid artery and brachial artery hemodynamics in nine normal subjects and 10 subjects with sustained essential hypertension. In hypertensive subjects, brachial artery blood flow and forearm vascular resistance were in the normal range while carotid artery blood flow and carotid artery resistance were decreased and increased, respectively. The most important findings were the changes in the internal caliber of large arteries. Although the brachial and carotid artery diameters of hypertensive subjects were measured for the same level of mean arterial pressure, brachial artery diameter was significantly increased and carotid artery diameter was strictly normal as compared with values found in normal subjects. To assess whether carotid artery circulation could influence the baroreceptor reflex response to arteriolar vasodilation, carotid artery and brachial artery hemodynamics were measured in immediate succession in normotensive and hypertensive subjects before and after oral administration of cadralazine, a dihydralazine derivative. After cadralazine treatment, carotid artery tangential tension decreased in hypertensive subjects, and the changes were significantly correlated to the Increase in heart rate. A similar correlation was found in normal subjects, but it was reset toward higher heart rates. These results indicate that the carotid artery does not behave like the brachial artery in response to a chronic increase in blood pressure. This behavior indicates intrinsic alterations of the arterial wall and might be involved in the resetting of the carotid baroreceptor reflex. Carotid artery circulation could play a role in hypertension by modulating the carotid baroreceptor mechanisms involved in the response to drug-induced arteriolar vasodilation. Whether these findings represent a primary or a secondary event remains to be determined. (CCA) and the brachial artery (BA) to a chronic elevation of blood pressure may differ according to the regional circulation. Using a noninvasive pulsed Doppler system, we evaluated arterial diameter and blood flow of the straight superficial arteries. We measured simultaneously CCA and BA hemodynamics in normal and hypertensive subjects to assess the effect of essential hypertension on circulation in these arteries.The decrease in blood pressure and vascular resistance observed after treatment with dihydralazine or its derivative, cadralazine, was related to the dilation of small arteries, as has been shown extensively for dihyFrom the Diagnosis Center and the Hypertension Research Center, Broussais Hospital, Paris, France.Address for reprints: Prof. M. Safar, Centre de Diagnostic, H6pi-tal Broussais,

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Section: Methodssupporting

confidence: 69%

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confidence: 80%

Hemodynamics of the carotid artery after vasodilation in essential hypertension.

et al. 1988

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“…Blood pressure and heart rate were determined automatically, every minute, by an oscillometric recorder (Dynamap, model 845, Critikon, Tampa, Florida, USA) on the left arm. 25 Body fat distribution and endothelium G Arcaro et al…”

Section: Haemodynamic Evaluationsmentioning

confidence: 99%

Body fat distribution predicts the degree of endothelial dysfunction in uncomplicated obesity

et al. 1999

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OBJECTIVE: To ascertain in obesity the role of body fat distribution (the strongest predictor of morbility and mortality in obese subjects) in determining the degree of endothelial dysfunction, an early marker of atherosclerotic disease. SUBJECTS: 18 premenopausal women with uncomplicated obesity excluding other cardiovascular risk factors and 12 age-matched slim healthy women. MEASUREMENTS: Endothelium-dependent vasodilation, studied as diameter variation in response to an increase in shear-stress, was evaluated in the right common femoral artery of obese and slim subjects by a non invasive approach and compared to glyceril-trinitrate vasodilation. To characterize better the vascular functional andaor structural properties, we studied the arterial wall distensibility by an echo-tracking system. Adipose tissue regional distribution was determined by computerised axial tomography. RESULTS: The endothelium-dependent vasodilation was signi®cantly impaired in obese subjects (P`0.005 versus non-obese subjects) while glyceril-trinitrate vasodilation and arterial distensibility were similar in the two groups. In our obese subjects endothelial-dependent vasodilation was inversely correlated to body fat distribution (visceralasubcutaneous adipose tissue ratio: r À0.624, P 0.0058). In contrast, metabolic parameters (except C-peptide response during oral glucose tolerance test (OGTT): r À0.587, P 0.01), blood pressure values and body weight did not correlate with the endothelial function. CONCLUSION: Uncomplicated obesity per se is characterised by an alteration of the endothelial function; the degree of this vascular damage is predicted by body fat distribution independently of body weight and metabolic and other haemodynamic parameters, and correlates with an index of insulin secretion.

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“…After at least 20 minutes' rest in a room with constant temperature (23 ± 1°C), the test was performed every time in basal condition and during an adrenergic stimulation (mental stress: consisting of complex mathematical calculations to execute in a short time period). Arterial pressure and heart rate were measured in the same times by means of a previously validated automatic device (Dinamap 845XT, Critikon, Johnson & Johnson, Tampa, FL, USA) [13].…”

Section: Methodsmentioning

confidence: 99%

The effects of thromboxane A2 inhibition (Picotamide) and angiotensin II receptor blockade (Losartan) in primary Raynaud's phenomenon

Pancera

Sansone

Secchi

et al. 1997

Journal of Internal Medicine

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Abstract. Pancera P, Sansone S, Secchi S, Covi G, Lechi A (University of Verona, Verona, Italy). The effects of thromboxane A2 inhibition (Picotamide) and angiotensin II receptor blockade (Losartan) in primary Raynaud's phenomenon. J Intern Med 1997; 242: 373-6. Objectives.To assess the role of thromboxane A2 and of angiotensin II in patients with primary Raynaud's phenomenon. Design. After an eight-day run-in period, the patients were enrolled in a single-blind, cross-over, study. Setting. Patients were examined at the Ambulatory for Microcirculatory Diseases of the Clinic of Internal Medicine, University Hospital, Verona. Subjects. Fifteen subjects affected by primary Raynaud's phenomenon were included. Main outcome measures. A piezoelectric plethysmography to evaluate the distensibility of the digital arteries as the ratio between peak time (PT) and total time (TT), and an oscillometric blood pressure recorder were used after the run-in period, and after a two-week course of picotamide (300 mg b.i.d., i.e. two times daily) or losartan (12.5 mg once daily) with an interval of a week of placebo between the active treatments. The tests were performed after every treatment in basal condition and during mental stress. The patients reported in a diary the number and the severity (from 0 to 4 +) of the vasospastic crises.Results. The change in TP/TT ratio appeared statistically significant only after losartan treatment, both in basal condition and during mathematical stress. Both pharmacological treatments, with respect to placebo, showed an improvement of the scores, derived from the number and severity of vasospastic attacks, but only the therapy with losartan determined a statistically significant improvement. Conclusions. The inhibition of the type 1 receptor for angiotensin II seems highly effective in the reduction of the vasospastic crises in the subjects with primary Raynaud's phenomenon. According to our experience, losartan could be used more extensively in the treatment of these patients besides arterial hypertension.

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Clinical evaluation of Dinamap 845 automated blood pressure recorder.

Cited by 128 publications

References 5 publications

Hemodynamics of the carotid artery after vasodilation in essential hypertension.

Hemodynamics of the carotid artery after vasodilation in essential hypertension.

Body fat distribution predicts the degree of endothelial dysfunction in uncomplicated obesity

The effects of thromboxane A2 inhibition (Picotamide) and angiotensin II receptor blockade (Losartan) in primary Raynaud's phenomenon

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