Clinical and Radiologic Features of Pulmonary Edema

Gluecker, Thomas M.; Capasso, Patrizio; Schnyder, P.; Gudinchet, F.; Schaller, Marie-Denise; Revelly, Jean-Pierre; Chioléro, René; Vock, Peter; Wicky, Stéphan

doi:10.1148/radiographics.19.6.g99no211507

Cited by 282 publications

(151 citation statements)

References 68 publications

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“…As it is common in the current literature, we are going to outline the plain radiological and CT appearances according to the pathological stage of the disease, even if a certain grade of overlap can be expected [47][48][49].…”

Section: Imaging Findings In Ardsmentioning

confidence: 99%

“…This observation suggests that atelectasis is an important factor in the inhomogeneous regional distribution of ARDS. Furthermore, this gravitational pattern can be helpful to exclude concomitant lung infections, as dependent atelectasis is more common in early ARDS without pneumonia [49,52].…”

Section: Imaging Findings In Ardsmentioning

confidence: 99%

“…Atypical ARDS, characterised by predominantly anterior consolidations in supine decubitus, can be observed in ,5% of patients during the first stage of the disease, probably due to regional differences in ventilation [49].…”

Section: Acute Respiratory Distress Syndrome | M Zompatori Et Almentioning

confidence: 99%

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Overview of current lung imaging in acute respiratory distress syndrome

2014

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Imaging plays a key role in the diagnosis and follow-up of acute respiratory distress syndrome (ARDS). Chest radiography, bedside lung ultrasonography and computed tomography scans can provide useful information for the management of patients and detection of prognostic factors. However, imaging findings are not specific and several possible differential diagnoses should be taken into account. Herein we will review the role of radiological techniques in ARDS, highlight the plain radiological and computed tomography findings according to the pathological stage of the disease (exudative, inflammatory and fibroproliferative), and summarise the main points for the differential diagnosis with cardiogenic oedema, which is still challenging in the acute stage. @ERSpublications ARDS diagnosis is still based on radiological criteria; the variety of these findings should be known and recognised

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Section: Imaging Findings In Ardsmentioning

confidence: 99%

Section: Imaging Findings In Ardsmentioning

confidence: 99%

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Overview of current lung imaging in acute respiratory distress syndrome

2014

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“…The second classification, contralateral pulmonary oedema, is oedema that occurs in a normal lung only as a result of an abnormality in the opposite lung. This occurs in people with congenital absence of a pulmonary artery, SwyerJames syndrome, pulmonary embolism, and lobectomy, among other conditions [8].…”

Section: Discussionmentioning

confidence: 99%

Localised right upper-lobe pulmonary oedema caused by extension of giant cell carcinoma to the mitral valve

Ono

Okada²,

Ando³

et al. 2011

BJR

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ABSTRACT. Giant cell carcinoma of the lung is a very rare primary malignant tumour and localised right upper-lobe pulmonary oedema is also unusual. We report a case of giant cell carcinoma, which invaded the left atrium through the left pulmonary vein and caused localised right upper-lobe pulmonary oedema. Giant cell carcinoma of the lung is a very rare primary malignancy associated with highly aggressive clinical behaviour and poor prognosis. To our knowledge very few CT images of patients with pulmonary giant cell carcinoma have been reported. Furthermore, localised right upper-lobe pulmonary oedema is also unusual. The case presented here exhibited clinical, radiographic and pathophysiological features of right upper-lobe pulmonary oedema. We report a case of localised right upperlobe pulmonary oedema caused by extension of giant cell carcinoma to the mitral valve. Case reportA 54-year-old man presented with an increasing cough and haemoptysis for 2 weeks. His past medical history included prior myocardial infarction and he underwent coronary artery bypass graft surgery 7 years ago. He had no history of pleuropulmonary disease. He was an alcoholic with a daily consumption of alcohol of more than 80 g during the past 10 years and had smoked for more than 10 pack-years (1 pack-year is an average of 1 pack of cigarettes smoked per day for a year).A chest radiograph showed complete opacification of the left hemithorax and elevation of the left diaphragm (not shown). Contrast chest thin-section CT images showed an ill-defined huge mass with extensive necrosis in the left lower lobe and multiple mediastinal and hilar lymph node enlargement (not shown). The left main bronchus was obstructed by a tumour with resultant left lung atelectasis. Moreover, the tumour had invaded the left atrium through the left pulmonary vein. Histopathological examination of a transbronchial biopsy showed non-small cell carcinoma (not shown).Although chemotherapy was administered to the patient, the tumour grew rapidly. A chest radiograph, which was obtained 1 month after the initial chest radiograph, revealed airspace shadowing confined to the right upper lung field (Figure 1). Contrast-enhanced thin-section CT images showed that the masses in the left lung, the left main bronchus and the left inferior pulmonary vein had increased in size (not shown). In addition, the tumour in the left atrium had also grown and the mitral valve dilated with invasion into the left ventricle (Figure 2). The CT images showed localised consolidation with air bronchogram in the right upper lobe, which was thought to be localised pulmonary oedema caused by mitral valve regurgitation owing to tumour progression to the left ventricle (Figure 3). In addition, metastatic right pulmonary small nodules and bilateral adrenal masses were observed on CT images (not shown).One week later the patient died of massive haemoptysis. The autopsy revealed that the left huge mass had invaded the left pulmonary vein, the left atrium and the left ventricle, and caused mitral val...

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“…Radiologically it presents with Kerley lines, peribronchial cuffing and, in severe cases, as central alveolar oedema without cardiomegaly on x ray chest. [7] Our main line of management was bag and mask ventilation and injection furosemide. Most patients who develop NPPE are managed with oxygen and furosemide, monitoring in the ICU, although some patient require intubation.…”

Section: Xray Chest Showing Features Of Pulmonary Edemamentioning

confidence: 99%

Patient developed NPPE in recovery room

Bshabshe¹,

Ashraf²

2017

Preprint

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Postoperative pulmonary edema is a rare life threatening complication associated with general anesthesia. Laryngospasm leads to forced inspiration generating large amount of negative intrathoracic pressure leading to negative pressure pulmonary edema. It usually develops immediately after extubation . This is a rare case report where patient developed NPPE in recovery room. In this paper we report a young fit adult who developed NPPE after an uneventful appendectomy in the post-operative period. The aim of this paper is to highlight the dramatic presentation of this rare condition in post-operative period and to remind that it can be lethal if immediate remedial steps are not taken. The anesthetist and paramedical staff in recovery room should be well aware of this condition.

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Clinical and Radiologic Features of Pulmonary Edema

Cited by 282 publications

References 68 publications

Overview of current lung imaging in acute respiratory distress syndrome

Overview of current lung imaging in acute respiratory distress syndrome

Localised right upper-lobe pulmonary oedema caused by extension of giant cell carcinoma to the mitral valve

Patient developed NPPE in recovery room

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