2007
DOI: 10.1016/j.echo.2007.05.017
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Circumferential Involvement of an Acute Type B Aortic Dissection: A Unique Case

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Cited by 4 publications
(3 citation statements)
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“…In addition, these valuable parameters can be further used as boundary conditions to establish more realistic computational fluid dynamic models and/ or in the study of finite element analysis on type B aortic dissection. Several extreme anatomical variations of type B aortic dissections, circumferential involvement of medial wall 22 as an example, can also be studied on these models with further modifications.…”
Section: Discussionmentioning
confidence: 99%
“…In addition, these valuable parameters can be further used as boundary conditions to establish more realistic computational fluid dynamic models and/ or in the study of finite element analysis on type B aortic dissection. Several extreme anatomical variations of type B aortic dissections, circumferential involvement of medial wall 22 as an example, can also be studied on these models with further modifications.…”
Section: Discussionmentioning
confidence: 99%
“…A free-floating or circumferential aortic dissection in which the true lumen is completely dissociated from the aortic wall is rare in TBAD. 26 In our experience, this is often associated with compression of the true lumen that leads to dynamic obstruction and the malperfusion syndrome. In this scenario, the patient would require early intervention, so it is not surprising that there were only seven patients with a free-floating true lumen included in the study.…”
Section: Journal Of Vascular Surgerymentioning
confidence: 91%
“…Gore [5] , Gore and Seiwert [6] and Gore and Hirst [7] theorized that the rupture of the elastin framework in the medial layer of the aorta is the primary pathological process in aortic dissection, and suggested that aortic dissection is the result of a congenital disorder of connective tissue which predominates in patients with aortic dissection who are less than 40 years old. Other investigators have argued that elastin rupture is a secondary pathological change caused by necrosis of smooth muscle cells or pathological accumulations in the matrix [8][9][10][11][12][13] .…”
Section: Discussionmentioning
confidence: 99%