Cigarette Smoking and Changes in Respiratory Findings

Comstock, George W.; Brownlow, Wilfred J.; Stone, Richard E.; Sartwell, Philip E.

doi:10.1080/00039896.1970.10667191

Cited by 55 publications

(17 citation statements)

References 7 publications

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“…Longitudinal studies are in line with the above crosssectional studies, showing that most intermittent symptoms (cough, phlegm and wheeze) decrease within 1-2 months after smoking cessation [21][22][23][24][25][26][27]. The prevalence of cough and wheeze decreases to that in nonsmokers, whereas the prevalence of phlegm remains slightly higher [21,25].…”

Section: Subject Without Chronic Respiratory Symptomssupporting

confidence: 62%

“…The prevalence of cough and wheeze decreases to that in nonsmokers, whereas the prevalence of phlegm remains slightly higher [21,25]. Furthermore, symptoms are also less likely to develop later in life if smokers without chronic symptoms quit smoking [28,29].…”

Section: Subject Without Chronic Respiratory Symptomsmentioning

confidence: 97%

“…In these studies, dyspnoea was defined as the feeling of "shortness of breath" or "having to stop for a breath while walking up a slight hill" or "walking with other people of the same age on the level ground" [25,27,28]. Another study suggested that 5 yrs of smoking cessation led to a small increase in dyspnoea when hurrying on the level or walking up a slight hill (41 to 52%) [21]. An increase in body weight, as often occurs during smoking cessation, might explain the increase in dyspnoea, but this was not investigated in this study.…”

Section: Subject Without Chronic Respiratory Symptomsmentioning

confidence: 99%

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The impact of smoking cessation on respiratory symptoms, lung function, airway hyperresponsiveness and inflammation

Willemse¹,

Postma²,

Timens³

et al. 2004

Eur Respir J

379

235

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Smoking is the main risk factor in the development of chronic obstructive pulmonary disease (COPD), and smoking cessation is the only effective treatment for avoiding or reducing the progression of this disease.Despite the fact that smoking cessation is a very important health issue, information about the underlying mechanisms of the effects of smoking cessation on the lungs is surprisingly scarce. It is likely that the reversibility of smoke-induced changes differs between smokers without chronic symptoms, smokers with nonobstructive chronic bronchitis and smokers with COPD. This review describes how these three groups differ regarding the effects of smoking cessation on respiratory symptoms, lung function (forced expiratory volume in one second), airway hyperresponsiveness, and pathological and inflammatory changes in the lung.Smoking cessation clearly improves respiratory symptoms and bronchial hyperresponsiveness, and prevents excessive decline in lung function in all three groups.Data from well-designed studies are lacking regarding the effects on inflammation and remodelling, and the few available studies show contradictory results. In chronic obstructive pulmonary disease, a few histopathological studies suggest that airway inflammation persists in exsmokers. Nevertheless, many studies have shown that smoking cessation improves the accelerated decline in forced expiratory volume in one second, which strongly indicates that important inflammatory and/or remodelling processes are positively affected.

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Section: Subject Without Chronic Respiratory Symptomssupporting

confidence: 62%

Section: Subject Without Chronic Respiratory Symptomsmentioning

confidence: 97%

Section: Subject Without Chronic Respiratory Symptomsmentioning

confidence: 99%

See 1 more Smart Citation

The impact of smoking cessation on respiratory symptoms, lung function, airway hyperresponsiveness and inflammation

Willemse¹,

Postma²,

Timens³

et al. 2004

Eur Respir J

379

235

View full text Add to dashboard Cite

show abstract

“…All this evidence indicates a causal and long lasting effect of smoking. Conversely, COMSTOCK et al [11] found that males who quit smoking showed a reduction in nasal symptoms over time which demonstrates that the effect of smoking is reversible to some extent.…”

Section: Discussionmentioning

confidence: 99%

“…Furthermore, COMSTOCK et al [11] found persistent nasal catarrh to be more prevalent among present smokers compared with non-smokers; smoking cessation, on the other hand, reduced this problem. These historical papers have since been supported by numerous studies.…”

Section: Smoking and Sinonasal Diseasementioning

confidence: 99%

Sinonasal inflammation in COPD: a systematic review

et al. 2012

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In this review, we demonstrate that patients with chronic obstructive pulmonary disease (COPD) frequently report sinonasal symptoms. Furthermore, we present evidence that smoking on its own can cause nasal disease and that, in COPD patients, nasal inflammation mimics that of the bronchi. All this evidence suggests that COPD-related sinonasal disease does exist and that smoking on its own rather than systemic inflammation triggers the condition. However, COPD-related sinonasal disease remains to be characterised in terms of symptoms and endoscopic findings. In addition, more studies are needed to quantify the negative impact of sinonasal symptoms on the quality of life in COPD patients. @ERSpublications A systematic review of sinonasal inflammation in chronic obstructive pulmonary disease

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Smoking and pulmonary function in five Solomon Island populations

Byerley

Weitz

Richards

1992

American J Phys Anthropol

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The relationship between smoking and lung function was examined in a large sample of healthy Solomon Island Melanesians and Polynesians as part of a multidisciplinary study carried out in 1985 and 1986. Comparisons are made between samples of smokers and non-smokers to determine if smoking is associated with a reduction in pulmonary function among males and females between 25 and 75 years of age. The results of this study indicate that cigarette smoking causes significant reductions in the pulmonary function of these populations. In addition, a clear difference in pulmonary function between males and females, as well as a general reduction in female FEV1 values, suggests that cooking smoke inhalation may be operating as a possible risk factor to the pulmonary health of women. Further research is needed to quantify exposure to smoke of both males and females and relate this measure to declines in pulmonary function.

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Cigarette Smoking and Changes in Respiratory Findings

Cited by 55 publications

References 7 publications

The impact of smoking cessation on respiratory symptoms, lung function, airway hyperresponsiveness and inflammation

The impact of smoking cessation on respiratory symptoms, lung function, airway hyperresponsiveness and inflammation

Sinonasal inflammation in COPD: a systematic review

Smoking and pulmonary function in five Solomon Island populations

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