Chronic Sialadenitis of the Submandibular Gland (so-called Küttner Tumor)

Yoshihara, Toshio; Kanda, Takashi; Yaku, Yuji; Kaneko, Toshio

doi:10.1016/s0385-8146(83)80036-4

Cited by 12 publications

(5 citation statements)

References 5 publications

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“…Chronic sclerosing sialadenitis affects almost exclusively the submandibular glands and is called Kuttner tumour in its advanced stage, as it presents clinically as a hard swelling indistinguishable from a tumour 101 . The disease can be bilateral.…”

Section: Known Tumour Entities With New Findingsmentioning

confidence: 99%

“…New concept of the nature of chronic sclerosing sialadenitis Chronic sclerosing sialadenitis affects almost exclusively the submandibular glands and is called Kuttner tumour in its advanced stage, as it presents clinically as a hard swelling indistinguishable from a tumour. 101 The disease can be bilateral. Patients are usually middle-aged or elderly and there is slight male predominance.…”

Section: Chronic Sclerosing Sialadenitis (Kuttner Tumour)mentioning

confidence: 99%

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Advances in salivary gland pathology

2007

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This review summarizes the new findings on salivary gland pathology under the following categories: immunohistochemistry; molecular genetics; newly recognized tumour types; known tumour entities with new findings; and progression of salivary gland tumours. In the application of immunohistochemistry, CD117 can aid in highlighting the luminal cell component of various salivary gland tumours, whereas p63 or maspin can aid in highlighting the abluminal cell component. A high Ki67 index remains the most useful marker to predict adverse outcome in salivary gland carcinoma. Specific chromosomal translocations are recognized in pleomorphic adenoma (with translocation involving PLGA1 or HMGA2 gene) and mucoepidermoid carcinoma (with MECT1-MAML2 gene fusion). Newly recognized entities include: sclerosing polycystic adenosis (with recent molecular evidence supporting its neoplastic nature), sclerosing mucoepidermoid carcinoma with eosinophilia, keratocystoma, adenoma with additional stromal component (lymphadenoma, lipoadenoma and adenofibroma), cribriform adenocarcinoma of the tongue and signet ring adenocarcinoma of minor salivary gland. Known tumour entities with new findings include: salivary duct carcinoma (with newly recognized mucinous, micropapillary and sarcomatoid variants), intraductal carcinoma (with controversies in terminology), mucoepidermoid carcinoma (with newly proposed grading parameters and oncocytic variant), epithelial-myoepithelial carcinoma (with newly recognized morphological variants), small cell carcinoma (with most cases being related to Merkel cell carcinoma), extranodal marginal zone B-cell lymphoma (with specific chromosomal translocation) and chronic sclerosing sialadenitis (being a component of IgG4-related sclerosing disease). Progression of salivary gland tumours can take the form of malignant transformation of a benign tumour, progression from low-grade to high-grade carcinoma, dedifferentiation, or stromal invasion of an in situ carcinoma.

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Section: Known Tumour Entities With New Findingsmentioning

confidence: 99%

Section: Chronic Sclerosing Sialadenitis (Kuttner Tumour)mentioning

confidence: 99%

Advances in salivary gland pathology

2007

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“…Some mechanisms have been postulated, including sialolithiasis, secretory dysfunction with ductal inspissation, duct abnormalities, infectious agents, and an autoimmune reaction. 3,8 In the largest study on CSS, Seifert et al 9 showed that the findings in early and late CSS were similar to those in obstructive sialadenitis; however, the progression of the inflammatory process could not be explained by purely obstructive mechanisms. In a recent study to investigate the role of immunopathologic processes in the pathogenesis of CSS, Tiemann et al 10 found that there was an intimate relationship between the T cell-dominated inflammatory infiltrate and the acinar and duct cells, together with the frequent demonstration of monoclonal and oligoclonal populations of cytotoxic T cells and their histopathologic behavior.…”

Section: Discussionmentioning

confidence: 97%

“…1,3,7,8 It is a benign process of uncertain cause. Some mechanisms have been postulated, including sialolithiasis, secretory dysfunction with ductal inspissation, duct abnormalities, infectious agents, and an autoimmune reaction.…”

Section: Discussionmentioning

confidence: 99%

Chronic Sclerosing Sialadenitis of the Parotid Gland

Chou

Tiu

et al. 2005

Journal of Ultrasound in Medicine

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“…Además, el examen físico generalmente mostrará una glándula significativamente indurada y, ocasionalmente, una glándula pequeña debido a la contractura de la cicatriz. La enfermedad de larga duración puede producir una masa similar a un tumor que se ha denominado tumor de Küttner (13). Si bien la anamnesis y el examen físico pueden ser suficientes para dar a los pacientes este diagnóstico, se pueden obtener estudios de imágenes especiales que documenten signos consistentes con SSCR.…”

Section: Diagnóstico Y Tratamiento De La Sialoadenitis Submandibular ...unclassified

Sialoadenitis Submandibular Crónica. Una Revisión De La Literatura

Bastidas

Vidarte

Castrejón

et al. 2022

SVS

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Objetivo: el objetivo de este estudio es recopilar la información más relevante en la literatura con respecto a la etiología, diagnóstico y tratamiento de la sialoadenitis submandibular crónica. Material y métodos: utilizando los buscadores PubMed y ScienceDirect se descargaron en total 143 artículos referentes al tema, de los cuales sólo se incluyeron 20 para la realización de este estudio. Resultados: la sialolitiasis es la principal causa de sialoadenitis submandibular, caracterizada por dolor postprandial y edema glandular. El ultrasonido es el estudio de imagen inicial que se debería solicitar debido a su bajo costo y accesibilidad. El tratamiento se basa principalmente en la escisión quirúrgica de la glándula en conjunto con el lito. Conclusiones: la sialoadenitis submandibular crónica es una de las principales patologías glandulares que se origina de una sialoadenitis aguda que no remitió al tratamiento inicial instaurado. Esta revisión permitirá resumir su fisiopatología, diagnóstico y tratamiento.

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Chronic Sialadenitis of the Submandibular Gland (so-called Küttner Tumor)

Cited by 12 publications

References 5 publications

Advances in salivary gland pathology

Advances in salivary gland pathology

Chronic Sclerosing Sialadenitis of the Parotid Gland

Sialoadenitis Submandibular Crónica. Una Revisión De La Literatura

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