Chronic sclerosing sialadenitis (Küttner's tumor) of the submandibular salivary gland: Our experience of one case report

Abstract

Surgical excision of the submandibular salivary gland in patients with chronic sclerosing sialadenitis can be complicated because of hyalinosis of the surrounding soft tissues. Patients with autoimmune diseases suspected of having salivary gland neoplasms or granulomatous disease should be carefully investigated for chronic sclerosing sialadenitis to prevent potential harm.

Mikulicz’s disease and Küttner’s tumor as manifestations of IgG4-related diseases: a review of the literature

“…Küttner's tumor is characterized by asymptomatic bilateral swelling of the SMG. Some patients might experience recurrent pain, discharge, and swelling during eating [20]. The duration of symptoms is highly variable, ranging from less than 1 year to many years [21].…”

“…Küttner's tumor could be histopathologically diagnosed by strong lymphocytic infiltration and fibrosis in the SMG either with or without sialolith [22]. Moreover, salivary duct obstruction, salivary stasis, sialolithiasis and secretory dysfunction play a role in the etiology of KT [20]. Sialoliths and mucous plugs are found in 29% to 83% of the lesions of KT [23].…”

“…Histopathological examination of KT reveals more prominent lymphocytic infiltration, formation of lymphoid follicles, atrophy of glandular parenchyma, advanced hyalinization in the periductal area, and sclerosis. Squamous and goblet cell metaplasia may be detected in the ductal system [20].…”

“…Although specimens of KT contain relatively large numbers of lymphoid cells and should be differentiated from malignant lymphoma of the SMG, some cytopathological features of KT may result in misinterpretation. Therefore, it is not a reliable method for diagnosing KT [20,37]. The utility of the parotid gland in diagnosing IgG4-RD has not been reported so far.…”

“…In SS patients, atrophic changes are found in both the parotid and SMG [ 13 ]. Ultrasonic examination in KT reveals diffuse changes in the SMG in the form of multiple hypoechoic lesions, heterogeneous background and duct dilatation [ 20 , 35 ]. Therefore, for the ultrasonic diagnosis of MD and KT a comprehensive assessment of both submandibular and parotid salivary glands is needed.…”