Characteristics of 24-hour Ambulatory Blood Pressure Monitoring in a COVID-19 Survivor

Wasim, Daanyaal; Almé, B.; Jordal, Stina; Eagan, Tomas Mikal; Tadić, Marijana; Mancia, Giuseppe; Guttormsen, Anne Berit; Saeed, Sahrai

doi:10.2217/fca-2020-0235

Cited by 9 publications

(13 citation statements)

References 11 publications

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“…91 The most frequent arrhythmia in patients with COVID-19 is sinus tachycardia-a homeostatic response to any infection or inflammatory response whose mechanism is, however, not fully understoodthough atrial fibrillation and ventricular tachycardia are also observed. [93][94][95] Among the mechanisms causing this condition, electrolyte imbalance, lung damage, drug side effects and cell signaling cascades after viral infection stands out. 75 In addition, hypoxia due to ARDS is correlated with an increased risk of arrhythmia.…”

Section: Cardiac Pathophysiologymentioning

confidence: 99%

COVID‐19 pathophysiology and ultrasound imaging: A multiorgan review

Ito

Rodrigues

Hümmelgen

et al. 2022

J of Clinical Ultrasound

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COVID‐19 is a dynamic disease and may affect different tissues and organs as it progresses. Therefore, the impact generated by the disease in all its stages and organs requires a functional and versatile imaging technique able to detect particularities or artifacts dynamically. Ultrasonography fulfills all these requirements and exhibit several advantages relative to other imaging modalities, including portability, lower cost and biosafety. Throughout the COVID‐19 pandemic, ultrasonography displayed a crucial role in the triage, monitoring, indicating organ damages and enabling individualized therapeutical decisions in COVID‐19 patients. This review is dedicated to highlight the main pathological effects correlated with ultrasound changes caused by COVID‐19 in the lungs, heart and liver.

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Section: Cardiac Pathophysiologymentioning

confidence: 99%

COVID‐19 pathophysiology and ultrasound imaging: A multiorgan review

Ito

Rodrigues

Hümmelgen

et al. 2022

J of Clinical Ultrasound

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“…Heart rate tends to be higher in survivors of COVID-19, and our study results differ from literature data. The difference in clinical variables such as sympathetic system activation and cytokine storm may have caused this difference [ 17 ].…”

Section: Discussionmentioning

confidence: 99%

Evaluation of Aortic Elasticity Parameters in Survivors of COVID-19 Using Echocardiography Imaging

et al. 2022

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Objective: While SARS-CoV-2 primarily affects lung tissue, it may cause direct or indirect damage to the cardiovascular system and permanent damage may occur. Arterial stiffness is an early indicator of cardiovascular disease risk. The aim of our study was to establish the potential effects of SARS-CoV-2 on the vascular system evaluated by transthoracic echocardiographic (TTE) examination. Subjects and Methods: This study compared arterial stiffness between the survivors of COVID-19 and those without a history of COVID-19 infection. The difference in aortic diameter was examined using echocardiography. Results: The study included 50 patients who survived COVID-19 in the last 3 to 6 months and 50 age- and gender-matched healthy volunteers. In surviving COVID-19 patients, aortic diastolic diameter in cm ([3.1±0.2] vs. [2.9±0.1], p < 0.001), pulse pressure ([43.02±14.05] vs [35.74±9.86], p = 0.004), aortic distensibility ([5.61±3.57] vs [8.31±3.82], p < 0.001), aortic strain ([10.56±4.91] vs [13.88±5.86], p = 0.003), pulse pressure/stroke volume index ([1.25±0.47] vs [0.98±0.28], p = 0.001) and aortic stiffness index ([2.82±0.47] vs [2.46±0.45], p < 0.001) were statistically significant compared to the control group. Conclusion: SARS-CoV-2 may cause reduced or impaired aortic elasticity parameters linked to impaired arterial wall function in COVID-19 survivors compared with controls.

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“…Many other mechanisms may also explain a persistent elevated heart rate and blood pressure in COVID-19 patients, such as activation of the renin–angiotensin–aldosterone system and sympathetic system, endothelial dysfunction, vasculitis, ischemia, hyperreninemia secondary to volume overload and intravenous saline use, vasopressors use, and mechanical ventilation. 25 , 26 …”

Section: Discussionmentioning

confidence: 99%

“…Many other mechanisms may also explain a persistent elevated heart rate and blood pressure in COVID-19 patients, such as activation of the renin-angiotensin-aldosterone system and sympathetic system, endothelial dysfunction, vasculitis, ischemia, hyperreninemia secondary to volume overload and intravenous saline use, vasopressors use, and mechanical ventilation. 25,26 This retrospective study explored the significance of sinus tachycardia on the clinical course of patients with mild COVID-19 infection, who did not require oxygen support or HD/ICU admission. Cohort analysis of sinus tachycardia demonstrated statistical significance for sinus tachycardia to increased CRP, abnormal CXR findings and prolonged hospitalization.…”

Section: Discussionmentioning

confidence: 99%

The clinical implications of sinus tachycardia in mild COVID-19 infection: A retrospective cohort study

et al. 2021

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Objectives: This study aims to estimate the prevalence of sinus tachycardia in hospitalized patients with mild COVID-19 infection and to identify the clinical, radiological, and biological characteristics associated with sinus tachycardia. Methods: A retrospective cohort study was conducted on patients with mild COVID-19 infection and sinus tachycardia during hospitalization. Outcomes measured included incidences of venous thromboembolism, high-dependency/intensive care unit admission, laboratory parameters, and radiological findings. Results: A total of 236 COVID-19 positive patients admitted to Singapore General Hospital isolation general wards from 1 June 2020 to 30 June 2020 were included in this study. Ninety-seven (41.1%) patients had sinus tachycardia on or during their admission. All patients were monitored in general wards and discharged to community quarantine facilities. None required oxygen support or high-dependency/intensive care unit admission. Sinus tachycardia was associated with increased C-reactive protein level (odds ratio = 1.033, 95% confidence interval = 1.002–1.066), abnormal chest X-ray findings (odds ratio = 3.142, 95% confidence interval = 1.390–7.104), and longer hospitalization (odds ratio = 1.117, 95% confidence interval = 1.010–1.236). There was no significant statistical association between sinus tachycardia and incidences of venous thromboembolism. Conclusion: This study suggests that patients with mild COVID-19 infection and concurrent sinus tachycardia are more likely to have higher inflammatory marker levels, abnormal imaging, and prolonged hospitalization. However, no significant association between sinus tachycardia and thromboembolism is identified in mild COVID-19 infection.

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Characteristics of 24-hour Ambulatory Blood Pressure Monitoring in a COVID-19 Survivor

Cited by 9 publications

References 11 publications

COVID‐19 pathophysiology and ultrasound imaging: A multiorgan review

COVID‐19 pathophysiology and ultrasound imaging: A multiorgan review

Evaluation of Aortic Elasticity Parameters in Survivors of COVID-19 Using Echocardiography Imaging

The clinical implications of sinus tachycardia in mild COVID-19 infection: A retrospective cohort study

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