Changing trends in the casuistics of hepatic abscess

Sherman, J.D.; Robbins, Stanley L.

doi:10.1016/0002-9343(60)90203-5

Cited by 78 publications

(28 citation statements)

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“…just as has been shown in patients not t!iven immunosuppressive dru~s in whom liver abscesses nevertheless develop; under the latter circumstances, ascendin~ cholan~itis is found in about -10 per cent of cases. 17 Whether the secondary bacterial invasion of the homografts was throu~h the biliary tract or the blood supply (particularly the portal vein) or both is not known in human patients and could not be determined in laboratorv studies of pit! and dot!…”

Section: I/()slimentioning

confidence: 99%

Infections in Recipients of Liver Homografts

Fulginiti¹,

Scribner²,

Groth³

et al. 1968

N Engl J Med

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Seventeen patients received liver homografts between 1963 and May, 1968. The eight treated before July, 1967, died within 34 days; seven had progressive infections with gram-negative bacilli. Candida albicans and cytomegalovirus. The infections were similar to but more fulminating than those after renal homotransplantation.In nine later cases. there was more discriminating donor selection. improved immunosuppression. and better organ preservation. In the first five of these nine patients. all infants, lobar hepatic gangrene

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Section: I/()slimentioning

confidence: 99%

Infections in Recipients of Liver Homografts

Fulginiti¹,

Scribner²,

Groth³

et al. 1968

N Engl J Med

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“…The etiologies and pathogenesis of liver abscesses have changed significantly over the past several decades [1][2][3][10][11] . Recent reports suggest that pyogenic liver abscess might be a warning indicator of silent colonic cancer [4,5,[12][13][14] , therefore, patients with liver abscess but without apparent etiologies, as in Case 1 of this report, should have further examination.…”

Section: Discussionmentioning

confidence: 99%

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mentioning

confidence: 99%

Pyogenic liver abscess associated with large colonic tubulovillous adenoma

Lai¹,

Chan

Peng³

et al. 2006

WJG

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tract disease or intra-abdominal infections including cholecystitis, suppurative cholangitis, suppurative pylephlebitis, appendicitis, diverticulitis and peritonitis [1][2][3] . Recently, colorectal cancer without obvious infection was considered as one of the uncommon etiologies of liver abscess [4,5] . Furthermore, liver abscess was reported as the presenting manifestation of colonic tubulovillous adenoma [6] . However, there are a growing number of reports suggesting a relationship between diabetes mellitus (DM) and Klebsiella pneumonia (KP) liver abscess which indicate that a deficiency in the hosts' defense mechanism could be the etiology of this liver infection [7][8][9] . We hereby report two cases of pyogenic liver abscess both of which were found to have colonic tubulovillous adenoma two to three weeks later after the diagnosis of liver abscess. The association of pyogenic liver abscess and colonic tubulovillous adenoma and the implications of these two cases are discussed. CASE REPORT CASE 1A 67-year-old man, who was referred from a local hospital, was admitted to our hospital because of fever, chill, and right upper abdominal pain for seven days. On physical examination, his initial body temperature was 37.9 o C and blood pressure was 124/67 mmHg. Light palpation of the abdomen revealed tenderness and mild rebounding pain on epigastric and right upper areas, but no abdominal mass was felt. Significant laboratory data included a leukocyte count of 26,330/mL (normal range = 4000-10,400/mL ), a hemoglobin of 9.5 gm/dL (normal range = 12-16 gm/dL), an aspartate aminotransferase (AST) of 73 IU/L (normal range = 5-34 IU/L), an alanine aminotranferase (ALT) of 79 IU/L (normal range = 0-40 IU/L), a fasting blood sugar of 118 mg/dl (normal range = 70-110 mg/dL), a total bilirubin of 1.03 mg/dL (normal range = 0.2-1.3 mg/dL), a C-Reactive Protein (CRP) of 17.02 mg/dL (normal <0.8 mg/dL), and a carcinoembryonic antigen (CEA) level of 0.33 ng/dL (normal <5 ng/dL).Abdominal ultrasonography (US) and computed tomography (CT) showed a 4-cm liver abscess in left lobe of liver ( Figure 1A). After three sets of blood cultures were completed and broad-spectrum antimicrobial therapy was initiated, CT-guided percutaneous drainage of the liver abscess was performed and pus liked material was aspirated and sent for bacterial culture. However, blood and pus were all negative for bacterial growth. As antimicrobial treatment was continued, the patient's clinical condition improved rapidly and fever subsided four days CASE REPORT P yo g en ic liver absc ess a s s o c i a t e d w i t h l a r g e c o l o n i c tubulovillous adenomaHsueh-Chou Lai, Cho-Yu Chan, Cheng-Yuan Peng, Chih-Bin Chen, Wen-Hsin Huang AbstractPyogenic liver abscesses usually occur in association with a variety of diseases. Rarely, liver abscess has been reported as the presenting manifestation of colonic tubulovillous adenoma. We report two cases of pyogenic liver abscess without hepatobiliary disease or other obvious etiologies except that one had a history ...

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“…Appendicitis was the etiologic agent in 34.2%. An update of hepatic abscesses by Sherman and Robbins in 1958 [8] revealed the most frequent antecedent cause to be cholangitis secondary to obstruction of the biliary tree. Recent reviews stress the importance of anaerobic organisms as a cause of intrahepatic abscess [9,10].…”

Section: Discussionmentioning

confidence: 99%