Abstract:Heat is the primary weather-related cause of death in the United States. Increasing heat and humidity, at least partially related to anthropogenic climate change, suggest that a long-term increase in heat-related mortality could occur. We calculated the annual excess mortality on days when apparent temperatures--an index that combines air temperature and humidity--exceeded a threshold value for 28 major metropolitan areas in the United States from 1964 through 1998. Heat-related mortality rates declined signif… Show more
“…Across the study area, the nolag-time prediction model in the hot weather group was usually more significant than other lag-time models. This finding is consistent with that of previous studies (e.g., Dessai 2002;Rainham and Smoyer-Tomic 2003;Sheridan and Kalkstein 2004), although other studies identified a 1-or 2-day lag between the mortality response and a heat event (e.g., Rogot and Padgett 1976;Davis et al 2003). For the rest of the weather groups (e.g., pollution-related or cold), most of the prediction models were more significant with 1-day lag or no lag time between the deaths and the environmental factors.…”
Section: Elevated Mortality Predictionssupporting
confidence: 93%
“…In hot and cold weather groups, 15:00 local standard time (LST) temperature was used to sort the data from lowest to highest; then the days were put into ranking groups with a roughly equal number of days in each ranking group. The afternoon was chosen as the time to take the temperature reading because the analysis revealed this to be the time when the strongest relationships with elevated mortality exist; this finding is consistent with previous studies (e.g., Smoyer et al 1999;Davis et al 2003). In air pollutionrelated weather groups, an air pollutant was used to rank the data for that pollutant-related weather group; for example, O 3 was used to rank the data for O 3 -related weather group.…”
This paper forms the second part of an introduction to a synoptic weather typing approach to assess differential and combined impacts of extreme temperatures and air pollution on human mortality, focusing on future estimates. A statistical downscaling approach was used to downscale daily five general circulation model (GCM) outputs (three Canadian and two US GCMs) and to derive six-hourly future climate information for the selected cities (Montreal, Ottawa, Toronto, and Windsor) in south-central Canada. Discriminant function analysis was then used to project the future weather types, based on historical analysis defined in a companion paper (Part I). Future air pollution concentrations were estimated using the within-weather-type historical simulation models applied to the downscaled future GCM climate data. Two independent approaches, based on (1) comparing future and historical frequencies of the weather groups and (2) applying within-weather-group elevated mortality prediction models, were used to assess climate change impacts on elevated mortality for two time windows (2040-2059 and 2070-2089). Averaging the five GCM scenarios, across the study area, heat-related mortality is projected to be more than double by the 2050s and triple by the 2080s from the current condition. Cold-related mortality could decrease by about 45-60% and 60-70% by the 2050s and the 2080s, respectively. Air pollution-related mortality could increase about 20-30% by the 2050s and 30-45% by the 2080s, due to increased air pollution levels projected with climate change. The increase in air pollution-related mortality would be largely driven by increases in ozone effects. The population acclimatization to increased heat was also assessed in this paper, which could reduce future heat-related mortality by 40%. It is most likely that the estimate of future extreme temperature-and air pollutionrelated mortality from this study could represent a bottomline figure since many of the factors (e.g., population growth, age structure changes, and adaptation measures) were not directly taken into account in the analyses.
“…Across the study area, the nolag-time prediction model in the hot weather group was usually more significant than other lag-time models. This finding is consistent with that of previous studies (e.g., Dessai 2002;Rainham and Smoyer-Tomic 2003;Sheridan and Kalkstein 2004), although other studies identified a 1-or 2-day lag between the mortality response and a heat event (e.g., Rogot and Padgett 1976;Davis et al 2003). For the rest of the weather groups (e.g., pollution-related or cold), most of the prediction models were more significant with 1-day lag or no lag time between the deaths and the environmental factors.…”
Section: Elevated Mortality Predictionssupporting
confidence: 93%
“…In hot and cold weather groups, 15:00 local standard time (LST) temperature was used to sort the data from lowest to highest; then the days were put into ranking groups with a roughly equal number of days in each ranking group. The afternoon was chosen as the time to take the temperature reading because the analysis revealed this to be the time when the strongest relationships with elevated mortality exist; this finding is consistent with previous studies (e.g., Smoyer et al 1999;Davis et al 2003). In air pollutionrelated weather groups, an air pollutant was used to rank the data for that pollutant-related weather group; for example, O 3 was used to rank the data for O 3 -related weather group.…”
This paper forms the second part of an introduction to a synoptic weather typing approach to assess differential and combined impacts of extreme temperatures and air pollution on human mortality, focusing on future estimates. A statistical downscaling approach was used to downscale daily five general circulation model (GCM) outputs (three Canadian and two US GCMs) and to derive six-hourly future climate information for the selected cities (Montreal, Ottawa, Toronto, and Windsor) in south-central Canada. Discriminant function analysis was then used to project the future weather types, based on historical analysis defined in a companion paper (Part I). Future air pollution concentrations were estimated using the within-weather-type historical simulation models applied to the downscaled future GCM climate data. Two independent approaches, based on (1) comparing future and historical frequencies of the weather groups and (2) applying within-weather-group elevated mortality prediction models, were used to assess climate change impacts on elevated mortality for two time windows (2040-2059 and 2070-2089). Averaging the five GCM scenarios, across the study area, heat-related mortality is projected to be more than double by the 2050s and triple by the 2080s from the current condition. Cold-related mortality could decrease by about 45-60% and 60-70% by the 2050s and the 2080s, respectively. Air pollution-related mortality could increase about 20-30% by the 2050s and 30-45% by the 2080s, due to increased air pollution levels projected with climate change. The increase in air pollution-related mortality would be largely driven by increases in ozone effects. The population acclimatization to increased heat was also assessed in this paper, which could reduce future heat-related mortality by 40%. It is most likely that the estimate of future extreme temperature-and air pollutionrelated mortality from this study could represent a bottomline figure since many of the factors (e.g., population growth, age structure changes, and adaptation measures) were not directly taken into account in the analyses.
“…These "thresholds" are about 17-18°C in Northern and Central Europe, 22-23°C in Southern Europe, 25°C on the Eastern Coast of the United States, and 26-29°C in Australia and South-East Asia. The adverse effects of increased temperatures can be prolonged for many days (so-called lagged effects) (Braga and Zanobetti 2002;Conti et al 2005;Curriero et al 2002;Davis et al 2003a;Davis et al 2003b;Dessai 2002;Donaldson et al 2001;Donaldson et al 2003;Gosling et al 2007;Gouveia et al 2003;Hajat et al 2005;Huynen et al 2001;Keatinge et al 2000;Michelozzi et al 2005;O'Neill et al 2003;Paldy et al 2005;Pattenden et al 2003;Sartor et al 1995;Vandentorren et al 2004). In addition, there is evidence suggesting that colder than normal temperatures can increase mortality (Carson et al 2001;Doyon et al 2008;Goodwin 2007;Gouveia et al 2003;Kovats et al 1998;Martens 1998;McMichael et al 2006), although these effects appear to be delayed for as many as two weeks into the future (Braga and Zanobetti 2002;Gouveia et al 2003;Huynen et al 2001;Pattenden et al 2003).…”
Section: The Temperature-mortality Relationshipmentioning
“…A smooth term with a maximum representing the MDT with a lag of 0 to 10 days (MDT (0-10) ) was used in the subsequent generalized additive model, along with smooth terms for MDRH with a lag of 0 to 10 days (MDRH (0-10) ), average DTGSR with the same lag period (DTGSR (0)(1)(2)(3)(4)(5)(6)(7)(8)(9)(10) ) and MDWS with the same lag period (MDWS (0-10) ). Same-day rainfall (square-root-transformed) was also included in all models based on the hypothesis that heavy rain would deter people from going to hospital.…”
Section: Hot Seasonmentioning
confidence: 99%
“…[1][2][3][4][5][6][7][8][9] Although there is evidence of an association between outdoor temperature and mortality rates, the interplay between the two remains only partially understood in Asia because of a lack of databases with comprehensive, comparable data for most urban communities in low-and middle-income settings. 10,11 Evidence from European and American cities suggests that when outdoor temperatures are unusually high, there is a rise in hospital admissions 10,[12][13][14][15] for respiratory ailments, renal diseases, 12 and infectious diseases (both vector-borne and foodborne) and cerebrovascular accidents, including subarachnoid haemorrhage 13 and transient ischaemic attacks.…”
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