Changes in Respiratory Dead Space During Halothane, Cyclopropane, and Nitrous Oxide Anesthesia

Askrog, V; PENDER, JOHN W.; Smith, Theodore C.; Eckenhoff, James E.

doi:10.1097/00000542-196405000-00013

Cited by 47 publications

(16 citation statements)

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“…2) is probably due in part to the prevalence of some degree of chronic respiratory disease in older patients. We have been unable to confirm the progressive increase in physiological deadspace which was suggested by Thornton (1960) and unequivocally demonstrated by Askrog et al (1964) during artificial ventilation with a variety of anaesthetic agents. Their results spanned 150 minutes, which was probably sufficiently long to show changes which were not revealed in the much shorter time covered by our own observations (table III).…”

Section: Discussioncontrasting

confidence: 58%

“…Theye and Tuohy (1964a) have studied changes in Po 2 during anaesthesia and did not observe any pattern of change in alveolararterial Po 2 gradients with time in individual patients. Askrog et al (1964) have reported serial changes during anaesthesia and, although they do not define the limits within which they controlled the concentration of oxygen in the inspired gas, their results, prolonged over 150 minutes, did not show progressive falls of arterial Po 2 of the type reported by Bendixen and his colleagues. Additional evidence has now been obtained by Sykes, Young and Robinson (1965).…”

Section: Discussionmentioning

confidence: 86%

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Factors influencing the arterial oxygen tension during anaesthesia with artificial ventilation. 1965

Nunn¹,

Bergmann²,

Coleman³

1998

British Journal of Anaesthesia

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Section: Discussioncontrasting

confidence: 58%

Section: Discussionmentioning

confidence: 86%

Factors influencing the arterial oxygen tension during anaesthesia with artificial ventilation. 1965

Nunn¹,

Bergmann²,

Coleman³

1998

British Journal of Anaesthesia

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“…The F ratio of these mean squares is significant for a = 0.01, demonstrating a nonzero component of variation between patients. The mean square estimate of the variance within patients is 6.0, and the mean square estimate of the additional component of variance between patients is 1.7.…”

Section: Resultsmentioning

confidence: 97%

Variation in PCO2 between Arterial Blood and Peak Expired Gas during Anesthesia

Raemer

Francis

Philip

et al. 1983

Anesthesia & Analgesia

100

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gas during anesthesia. Anesth Analg 1983;62:1065-9.with maximum measurement of Pam2 often over-or underestimated Paco2.Mean estimated Paco2 from calibrated P(a-pe)co2 varied Variation in PCOZ between arterial blood and peak expired torr, Calibration of P(a-pe)co, based on a single initial Arterial PcOz (Pace,) can be continuously and noninvasively estimated by monitoring peak expired CO, tension (Ppeco,). The practice of calibrating the estimate by an initial measurement of Paco2 assumes that the difference in PCO, tension between arterial blood and expired gas P(ape)coz remains constant. We examined the stability of P(afrom -7.9-6.4 torr with extreme estimates and duration of anesthesia, variations in ventilation, blood pressure, Ppeco, or temperature. We conclude that estimation of Paco, by monitoring Ppeco2 is not invariably reliable. pe)co, during anesthesia in 15 patients undergoing major surgery. Mean P(a-peko2 values rangedfrom 0.8-7.9 forr

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“…16 saw a progressive increase in the difference over time in seven patients undergoing major general surgery when deliberate hypotension was induced, due to an increase in dead space. In our study there were no major haemodynamic changes.…”

Section: Discussionmentioning

confidence: 99%

Stability of the arterial to end-tidal carbon dioxide difference during anaesthesia for prolonged neurosurgical procedures

1995

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This study was undertaken to examine the variation of the arterial to One of the major treatment modalities used by anaesthetists for the management of raised intracmnial pressure (ICP) in neurosurgical patients is hyperventilation, which reduces arterial PaCO2. Hyperventilation to a PaCO2 of 25-30 mmHg is the mainstay of acute and subacute management of raised ICE It is well known that PaCO2 correlates inversely with cerebral arterial resistance. As a con-CAN J ANAESTH 19~5 ] 42: 6 / pp 498-503

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Changes in Respiratory Dead Space During Halothane, Cyclopropane, and Nitrous Oxide Anesthesia

Cited by 47 publications

References 0 publications

Factors influencing the arterial oxygen tension during anaesthesia with artificial ventilation. 1965

Factors influencing the arterial oxygen tension during anaesthesia with artificial ventilation. 1965

Variation in PCO2 between Arterial Blood and Peak Expired Gas during Anesthesia

Stability of the arterial to end-tidal carbon dioxide difference during anaesthesia for prolonged neurosurgical procedures

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