2011
DOI: 10.1016/j.ijcard.2011.08.034
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Changes in endothelial function in healthy volunteers during soccer World Cup 2010 games

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Cited by 2 publications
(2 citation statements)
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References 14 publications
(16 reference statements)
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“…Stress resulting in increased sympathetic nervous activity and coagulability contributes to an increased risk for transformation of a non-vulnerable atherosclerotic plaque into a plaque, which is susceptible for disruption accompanied by thrombogenic stimulus and can induce further vasoconstriction as well as increased coagulability, resulting in acute coronary syndrome including MI 14 , 17 . This was supported by a study showing that watching the FIFA WC 2010 between Spain and the Netherlands, Spanish fans had significant higher levels of testosterone and cortisol compared with control days 17 , 37 and by another study showing changes in endothelial function in healthy volunteers during the FIFA soccer WC 2010 36 .…”
Section: Discussionmentioning
confidence: 77%
See 1 more Smart Citation
“…Stress resulting in increased sympathetic nervous activity and coagulability contributes to an increased risk for transformation of a non-vulnerable atherosclerotic plaque into a plaque, which is susceptible for disruption accompanied by thrombogenic stimulus and can induce further vasoconstriction as well as increased coagulability, resulting in acute coronary syndrome including MI 14 , 17 . This was supported by a study showing that watching the FIFA WC 2010 between Spain and the Netherlands, Spanish fans had significant higher levels of testosterone and cortisol compared with control days 17 , 37 and by another study showing changes in endothelial function in healthy volunteers during the FIFA soccer WC 2010 36 .…”
Section: Discussionmentioning
confidence: 77%
“…Pathophysiological, it has been hypothesized, that emitted stress hormones might directly affect endothelial, monocytic and platelet functions 17 , 36 . Stress resulting in increased sympathetic nervous activity and coagulability contributes to an increased risk for transformation of a non-vulnerable atherosclerotic plaque into a plaque, which is susceptible for disruption accompanied by thrombogenic stimulus and can induce further vasoconstriction as well as increased coagulability, resulting in acute coronary syndrome including MI 14 , 17 .…”
Section: Discussionmentioning
confidence: 99%