Cerebral Venous Thrombosis

Caplan, Louis R.

doi:10.1016/b978-1-4160-4721-6.50018-3

Cited by 5 publications

(2 citation statements)

References 121 publications

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“…However, if the rostral SSS is atretic or hypoplastic, substitute parasagittal venous channels develop and drain most of the frontal lobe 5,18) . Thus, an acute occlusion of such a parasagittal vein can cause a rise in the venous pressure and tissue pressure within its drainage territory, leading to a decrease in the blood supply and disruption of the blood-brain barrier, which eventually results in regional brain edema and venous infarction with or without a hemorrhagic transformation 2,3,12,16) . Venous infarction develops ill-defined hypodense lesions that do not conform to the arterial distribution on a CT scan.…”

Section: Discussionmentioning

confidence: 99%

“…Hemorrhages, mainly in the white matter, range from petechial to large, and surrounding cerebral edema is prominent. Clinical features include frequent headaches and seizures, in addition to focal deficits and disorders of consciousness 2,3) . The clinical course of venous infarction is usually slow and indolent until hemorrhagic transformation.…”

Section: Discussionmentioning

confidence: 99%

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Cerebral Venous Thrombosis Complicated by Hemorrhagic Infarction Secondary to Ventriculoperitoneal Shunting

Son

Park

2010

J Korean Neurosurg Soc

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While a delayed intracerebral hemorrhage at the site of a ventricular catheter has occasionally been reported in literature, a delayed hemorrhage caused by venous infarction secondary to ventriculoperitoneal shunting has not been previously reported. In the present case, a 68-year-old woman underwent ventriculoperitoneal shunting through a frontal burr hole, and developed a hemorrhagic transformation of venous infarction on the second postoperative day. This massive venous infarction was caused by bipolar coagulation and occlusion of a large paramedian cortical vein in association with atresia of the rostral superior sagittal sinus. Thus, to eliminate the risk of postoperative venous infarction, technical precautions to avoid damaging surface vessels in a burr hole are required under loupe magnification in ventriculoperitoneal shunting.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Cerebral Venous Thrombosis Complicated by Hemorrhagic Infarction Secondary to Ventriculoperitoneal Shunting

Son

Park

2010

J Korean Neurosurg Soc

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Severe Headache and Hypertension due to Cerebral Venous Sinus Thrombosis in a Patient With Cervical Spinal Cord Injury: A Case Report

Cha

Kee

Moon

et al. 2022

Korean J Neurotrauma

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Diagnosis and Management of Mixed Transcortical Aphasia Due to Multiple Predisposing Factors, including Postpartum and Severe Inherited Thrombophilia, Affecting Multiple Cerebral Venous and Dural Sinus Thrombosis: Case Report and Literature Review

Jianu

Dan

et al. 2021

Diagnostics

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Cerebral venous and dural sinus thrombosis (CVT) is an uncommon disease in the general population, although it is a significant stroke type throughout pregnancy and the puerperium. Studies describing this subtype of CVT are limited. Most pregnancy-associated CVT happen in late pregnancy, or more commonly in the first postpartum weeks, being associated with venous thrombosis outside the nervous system. Case presentation: The current study describes a case of multiple CVT in a 38-year-old woman with multiple risk factors (including severe inherited thrombophilia and being in the puerperium period), presenting mixed transcortical aphasia (a rare type of aphasia) associated with right moderate hemiparesis and intracranial hypertension. The clinical diagnosis of CVT was confirmed by laboratory data and neuroimaging data from head computed tomography, magnetic resonance imaging, and magnetic resonance venography. She was successfully treated with low-molecular-weight heparin (anticoagulation) and osmotic diuretics (mannitol) for increased intracranial pressure and cerebral edema. At discharge, after 15 days of evolution, she presented a partial recovery, with anomic plus aphasia and mild right hemiparesis. Clinical and imaging follow-up was performed at 6 months after discharge; our patient presented normal language and mild right central facial paresis, with chronic left thalamic, caudate nucleus, and internal capsule infarcts and a partial recanalization of the dural sinuses.

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Cerebral Venous Thrombosis

Cited by 5 publications

References 121 publications

Cerebral Venous Thrombosis Complicated by Hemorrhagic Infarction Secondary to Ventriculoperitoneal Shunting

Cerebral Venous Thrombosis Complicated by Hemorrhagic Infarction Secondary to Ventriculoperitoneal Shunting

Severe Headache and Hypertension due to Cerebral Venous Sinus Thrombosis in a Patient With Cervical Spinal Cord Injury: A Case Report

Diagnosis and Management of Mixed Transcortical Aphasia Due to Multiple Predisposing Factors, including Postpartum and Severe Inherited Thrombophilia, Affecting Multiple Cerebral Venous and Dural Sinus Thrombosis: Case Report and Literature Review

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