“…It should be pointed out, however, that, as reported by Smith et al, the clinicopathological features of 2 SLE patients with vasculopathy in the brain were found to be, in one case, vasculitis with fibrinoid necrosis in a cerebellar vessel, whereas in the other case there was perivascular hemorrhage in small vessels containing fibrin thrombi, without any features of acute vasculitis [16]. In the brain hematoma of our patient, there was inflammatory cell accumulation mostly on the walls of small arteries, inducing rupture of the lamina elastica interna.…”