2014
DOI: 10.1159/000360014
|View full text |Cite
|
Sign up to set email alerts
|

Central Serous Chorioretinopathy

Abstract: The pathogenesis of central serous chorioretinopathy (CSC) is still not fully understood. The involvement of corticosteroids is undisputed, although their exact role has not been clarified; other parts of the underlying mechanism of CSC have been mainly elucidated by imaging techniques such as fluorescein and indocyanine green angiography. Even though most cases of CSC are self-limiting, severe as well as recurrent courses exist, and for these patients only a limited number of treatment options are available: … Show more

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
4
1

Citation Types

4
115
0
13

Year Published

2016
2016
2021
2021

Publication Types

Select...
7
2

Relationship

0
9

Authors

Journals

citations
Cited by 130 publications
(134 citation statements)
references
References 104 publications
4
115
0
13
Order By: Relevance
“…Acute CSC was diagnosed using FA, ICGA, and OCT in the light of previously reported findings. 13 No treatment was given to the patients during the first 6 months. The patients were treated with PDT after a minimum time of 6 months from the initial signs of the disease, if they did not show any sign of resolution or if any increase was detected in SRF.…”
Section: Methodsmentioning
confidence: 99%
“…Acute CSC was diagnosed using FA, ICGA, and OCT in the light of previously reported findings. 13 No treatment was given to the patients during the first 6 months. The patients were treated with PDT after a minimum time of 6 months from the initial signs of the disease, if they did not show any sign of resolution or if any increase was detected in SRF.…”
Section: Methodsmentioning
confidence: 99%
“…A variety of risk factors have been associated with CSC, including psychological stress, type A personality, hypertension, systemic steroid usage, pregnancy, and genetic susceptibility [3]. The exact pathogenesis of the disease is not fully understood, but abnormalities in the inner choroidal layers, such as venous congestion, ischemia and/or inflammation, lead to choroidal hyper permeability, secondary RPE damage, and serous detachment of the neural retina [3][4][5]. CSC is self-limited in the majority of patients, who usually retain excellent vision.…”
Section: Introductionmentioning
confidence: 99%
“…It has a benign and self-limiting nature, with a long-term recurrence rate of 30%. CSCR usually occur in males in 20-50 years [1][2][3][4][5][6].…”
Section: Introductionmentioning
confidence: 99%