2000
DOI: 10.1034/j.1600-0757.2000.2240107.x
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Cell biology of gingival wound healing

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Cited by 202 publications
(190 citation statements)
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“…However, dramatically increased numbers of myofibroblasts appear during the wound healing processes [Hakkinen et al, 2000]. It has been found that myofibroblast differentiation occurs in HGFs in vitro [Arora and McCulloch, 1999], and our results confirmed that upon TGF-β1 stimulation HGFs could differentiate into myofibroblasts.…”
Section: Discussionsupporting
confidence: 83%
“…However, dramatically increased numbers of myofibroblasts appear during the wound healing processes [Hakkinen et al, 2000]. It has been found that myofibroblast differentiation occurs in HGFs in vitro [Arora and McCulloch, 1999], and our results confirmed that upon TGF-β1 stimulation HGFs could differentiate into myofibroblasts.…”
Section: Discussionsupporting
confidence: 83%
“…This new matrix supports the osteogenic cells migration Lang et al (2003), stimulated by several molecules released during blood clotting and clot cleansing, such as PDGF, TGF-β Davies (2003) and fibroblast growth factor (FGF) Dimitriou et al (2005). Between the 7-10th day of healing, some of the fibroblasts present in the interface are transformed into myoblasts Häkkinen et al (2000) characterized by smooth muscle α-actin cytoplasmatic microfilaments, allowing them to generate contractile forces responsible for wound contraction Aukhil (2000) Biological activity regarding wound healing at the bone-dental implant interface concludes with the modeling and subsequent bone remodeling Sikavitsas et al (2001). Moreover, cell adhesion, cell migration and proliferation on surrounding tissues, and internal and external mechanical loads action modify the new tissue formation profile ( Figure 4).…”
Section: Mechanobiology Of the Bone-dental Implant Interfacementioning
confidence: 96%
“…First, the adhesion phenomena produced by cells fixation to a substrate Anselme (2000) activate chemical signaling Kasemo (2002) controlling cell proliferation and differentiation, as in platelet aggregation and activation stages Collen & Lijnen (1991). Then, the contraction exerted by cells moving over a substrate Häkkinen et al (2000) may cause the displacement of the attached fibrin fibers and their detachment from the implant surface Davies (2003;. This kind of event is present during fibroplasia and modeling stages where fibroblasts and osteogenic cells adhere to the fibrin network and begin to move through it in an attempt to colonize the implant surface.…”
Section: Mechanobiology Of the Bone-dental Implant Interfacementioning
confidence: 99%
“…The difference lies in the gingival wound where keratinocyte migrates directly to fibrinfibronectin matrix without contacting with collagen 26,27 . The wound healing in mouth mucosa is relatively faster, and scraped tissues are seldom forming in this case.…”
Section: Discussionmentioning
confidence: 99%
“…Fibroblast in adult gingival is located in string tissue papilla, and some of its components share with the fibroblast of fetal. There are several factors in saliva, such as growth factor and ion (magnesium and calcium), that function to decrease some material components that may influence the gingival healing 27 . The moisturization of mouth by saliva also functions to help heal wound.…”
Section: Discussionmentioning
confidence: 99%