Catheter induced spasm in aortocoronary vein grafts.

Abstract

(Fig. 1A). The patient did not complain of chest pain, and there were no electrocardiographic changes. Ten minutes after injecting 100 ,ug glyceryl trinitrate and 0-2 mg nifedipine in the pulmonary artery repeat angiography was performed. The narrowed segment in the vein had disappeared (Fig. 1B)

Prolonged catheter-induced coronary artery spasm mimicking fixed stenosis

“…Several local vasoconstrictor mechanisms have been postulated. They include a myogenic stimulus, i.e., mechanical irritation or stress receptor activation [5], and platelet aggregation at the catheter tip with release of vasoactive agents [6]. While often producing vasodilatation, angiographic dye has also been reported to cause coronary vasoconstriction [7,8].…”

Catheter-induced Vasospasm of Both the Right External Iliac and Right Coronary Arteries During Coronary Angiography

“…In accordance with the presence of mild coronary artery disease (CAD) in the LAD and LCx in this patient, she also fits the typical prototype of patients predisposed to coronary vasospasm as previously defined by provocative testing with ergonovine [19]. Catheter-induced vasospasm varies in accordance with catheter use and angiographer expertise and not infrequently accompanies diagnostic intervention, particularly with radial artery access, and during PCI of both native vessels and coronary artery bypass grafts, potentially resulting from mechanical stretch of the artery or denudation of the endothelium [7]. Coronary vasospasm may also result following rotational, and laser atherectomy stemming from vibratory-induced release of vasoactive mediators and heat generation, respectively [20].…”

“…Proposed mechanisms include mechanical irritation triggering a myogenic reflex [6,7], stress receptor activation [8], and platelet aggregation at the catheter tip with release of vasoactive agents [9]. Several underlying etiologies have been documented including hypercholesterolemia, ergot toxicity, exercise, L-thyroxin therapy, hyperosmolar contrast medium, nonsteroidal anti-inflammatory drugs, cigarette smoking, and alcohol [7,8,[10][11][12][13][14][15][16][17][18].…”

Rarely seen temporary subclavian vein spasm and twitching on the epicardial battery

“…Daha sonra yapılan venogramda belirgin venöz spazm görülmüş, nitrogliserin uygulanmasına da yanıt alınamayınca implantasyon yeri sağa kaydırılmış ve başarılı bir şekilde pacemaker Kardiyovasküler girişimler sırasında arter spazmları venöz spazmlara göre çok daha sık görülmektedir. Arter spazmlarının mekanizmasının miyojenik refleksi tetikleyen mekanik irritasyon, reseptör aktivasyonu ve vazoaktif ajanların salınımıyla kateter ucunda trombosit agregasyonu olduğu öne sürülmüştür [6][7][8][9][10] . Venöz spazmların mekanizması tam olarak bilinmemektedir, bazı araştırmacılar bunun kontrast maddenin kimyasal etkisi, iğne girişiminin mekanik etkisi veya kılavuz telinin yerleştirilmesiyle ilişkili olabileceğini ileri sürmüşlerdir 2,3 .…”